Nuclear expression of Gli-1 is predictive of pathologic complete response to chemoradiation in trimodality treated oesophageal cancer patients.

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Hedgehog inhibition mediates radiation sensitivity in mouse xenograft models of human esophageal adenocarcinoma.Role of GLI Transcription Factors in Pathogenesis and Their Potential as New Therapeutic Targets Hedgehog Signaling in Lung Cancer: From Oncogenesis to Cancer Treatment Resistance

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P2860

Nuclear expression of Gli-1 is predictive of pathologic complete response to chemoradiation in trimodality treated oesophageal cancer patients.

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Ailing W Scott

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Brian Weston

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David C Rice

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Dilsa Mizrak Kaya

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Dipen M Maru

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Donald Berry

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Elena Elimova

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Heath D Skinner

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Hironori Shiozaki

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Jeffrey H Lee

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P2860

The crosstalk of mTOR/S6K1 and Hedgehog pathways

Nonparametric Estimation from Incomplete Observations

In vitro propagation and transcriptional profiling of human mammary stem/progenitor cells

Inhibition of the hedgehog pathway in advanced basal-cell carcinoma.

Expression of DNMT1 and DNMT3a are regulated by GLI1 in human pancreatic cancer.

Complete pathologic response after neoadjuvant chemoradiotherapy for esophageal cancer is associated with enhanced survival.

Hedgehog signalling in cancer formation and maintenance.

A crucial requirement for Hedgehog signaling in small cell lung cancer

Complete response to neoadjuvant chemoradiotherapy in esophageal carcinoma is associated with significantly improved survival.

Clinical parameters model for predicting pathologic complete response following preoperative chemoradiation in patients with esophageal cancer.

P2888

bjc.2017.225

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648-655

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10.1038/BJC.2017.225

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Bin Liu

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1090829033

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chemoradiotherapy

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Nuclear expression of Gli-1 is predictive of pathologic complete response to chemoradiation in trimodality treated oesophageal cancer patients.

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P2860

P2860

Nuclear expression of Gli-1 is predictive of pathologic complete response to chemoradiation in trimodality treated oesophageal cancer patients.

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