Hepatocytes contribute to residual glucose production in a mouse model for glycogen storage disease type Ia.

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Flux analysis of inborn errors of metabolism.Intracellular lipids are an independent cause of liver injury and chronic kidney disease in non alcoholic fatty liver disease-like context

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Hepatocytes contribute to residual glucose production in a mouse model for glycogen storage disease type Ia.

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Hepatocytes contribute to resi ...... cogen storage disease type Ia.

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Hepatocytes contribute to resi ...... cogen storage disease type Ia.

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Hepatocytes contribute to resi ...... cogen storage disease type Ia.

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Hepatocytes contribute to resi ...... ycogen storage disease type Ia

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Albert K Groen

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Andreas Boss

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Arend Heerschap

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Brenda S Hijmans

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Elodie Mutel

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Fabienne Rajas

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Gilles Mithieux

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Henk Wolters

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Maaike H Oosterveer

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Maud Soty

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P2860

Starch binding domain-containing protein 1/genethonin 1 is a novel participant in glycogen metabolism

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Natural history of hepatocellular adenoma formation in glycogen storage disease type I

Starch Binding Domain-containing Protein 1 Plays a Dominant Role in Glycogen Transport to Lysosomes in Liver

Estimation of glucose carbon recycling in children with glycogen storage disease: A 13C NMR study using [U-13C]glucose.

Mechanism by which hyperglycemia inhibits hepatic glucose production in conscious rats. Implications for the pathophysiology of fasting hyperglycemia in diabetes.

Identification and characterization of a human cDNA and gene encoding a ubiquitously expressed glucose-6-phosphatase catalytic subunit-related protein.

Glucokinase and molecular aspects of liver glycogen metabolism.

Hepatic gluconeogenic fluxes and glycogen turnover during fasting in humans. A stable isotope study

Role of glycogen phosphorylase in liver glycogen metabolism.

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2042-2054

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scholarly article

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10.1002/HEP.29389

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English

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Terry G J Derks

Dirk-Jan Reijngoud

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2017-10-30T00:00:00Z

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28727166

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Hepatocytes contribute to residual glucose production in a mouse model for glycogen storage disease type Ia.

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P2860

P2860

Hepatocytes contribute to residual glucose production in a mouse model for glycogen storage disease type Ia.

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