Conditionally Immortal Slc4a11-/- Mouse Corneal Endothelial Cell Line Recapitulates Disrupted Glutaminolysis Seen in Slc4a11-/- Mouse Model.

about

Conditionally Immortal Slc4a11-/- Mouse Corneal Endothelial Cell Line Recapitulates Disrupted Glutaminolysis Seen in Slc4a11-/- Mouse Model.

Item

http://www.wikidata.org/entity/Q38696524

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

Conditionally Immortal Slc4a11 ...... een in Slc4a11-/- Mouse Model.

@en

type

Item

label

Conditionally Immortal Slc4a11 ...... een in Slc4a11-/- Mouse Model.

@en

prefLabel

Conditionally Immortal Slc4a11 ...... een in Slc4a11-/- Mouse Model.

@en

P1476

Conditionally Immortal Slc4a11 ...... Seen in Slc4a11-/- Mouse Model

@en

P2093

Diego G Ogando

http://www.w3.org/2001/XMLSchema#string

Edward T Kim

http://www.w3.org/2001/XMLSchema#string

Jason M Tenessen

http://www.w3.org/2001/XMLSchema#string

Joseph A Bonanno

http://www.w3.org/2001/XMLSchema#string

Moon-Jung Choi

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphate-activated glutaminase (GLS2), a p53-inducible regulator of glutamine metabolism and reactive oxygen species

Genetic analysis of two Indian families affected with congenital hereditary endothelial dystrophy: two novel mutations in SLC4A11

Borate transporter SLC4A11 mutations cause both Harboyan syndrome and non-syndromic corneal endothelial dystrophy

Autosomal recessive corneal endothelial dystrophy (CHED2) is associated with mutations in SLC4A11

Mutation analysis of the SLC4A11 gene in Indian families with congenital hereditary endothelial dystrophy 2 and a review of the literature

SV40 large T antigen targets multiple cellular pathways to elicit cellular transformation

Determination of metabolic flux ratios from 13C-experiments and gas chromatography-mass spectrometry data: protocol and principles.

Methods for studying metabolism in Drosophila

Glutaminolysis is Essential for Energy Production and Ion Transport in Human Corneal Endothelium.

Missense mutations in the sodium borate cotransporter SLC4A11 cause late-onset Fuchs corneal dystrophy.

P304

3723-3731

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1167/IOVS.17-21781

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Hongde Li

Wenlin Zhang

P577

2017-07-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28738416

http://www.w3.org/2001/XMLSchema#string

P921

endothelium

P932

5525555

http://www.w3.org/2001/XMLSchema#string