IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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Sentinels of the Type 2 Immune Response.The atopic march: Critical evidence and clinical relevance.

P2860

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P2860

IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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http://www.wikidata.org/entity/Q38705524

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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name

IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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P1476

IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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P2093

D E Smith

http://www.w3.org/2001/XMLSchema#string

F Roan

http://www.w3.org/2001/XMLSchema#string

H Han

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L K Johnston

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P J Bryce

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S F Ziegler

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P2860

Skin-derived TSLP triggers progression from epidermal-barrier defects to asthma

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

Factors associated with the development of peanut allergy in childhood

Gene polymorphism in Netherton and common atopic disease

Skin-specific expression of IL-33 activates group 2 innate lymphoid cells and elicits atopic dermatitis-like inflammation in mice

Exposure to food allergens through inflamed skin promotes intestinal food allergy through the thymic stromal lymphopoietin-basophil axis.

Mast cells are required for experimental oral allergen-induced diarrhea

Filaggrin loss-of-function mutations are associated with food allergy in childhood and adolescence

Peanut allergy: effect of environmental peanut exposure in children with filaggrin loss-of-function mutations.

The H1 histamine receptor regulates allergic lung responses.

P2888

mi.2017.61

P31

scholarly article

P356

10.1038/MI.2017.61

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P577

2017-06-28T00:00:00Z

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1090275086

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28656964

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P932

5745299

http://www.w3.org/2001/XMLSchema#string

IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

about

P2860

P2860

IL-33 promotes gastrointestianl allergy in a TSLP-independent manner.

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