The ACE2-Ang (1-7)-Mas receptor axis attenuates cardiac remodeling and fibrosis in post-myocardial infarction

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The ACE2-Ang (1-7)-Mas receptor axis attenuates cardiac remodeling and fibrosis in post-myocardial infarction

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The ACE2-Ang (1-7)-Mas recepto ...... in post-myocardial infarction

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The ACE2-Ang (1-7)-Mas recepto ...... in post-myocardial infarction

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The ACE2-Ang (1-7)-Mas recepto ...... in post-myocardial infarction

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The ACE2-Ang (1-7)-Mas recepto ...... in post-myocardial infarction

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Difei Shen

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Hui Li

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Juan Wang

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Liping Guo

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Suxia Han

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Wen He

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Yin Zhang

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P2860

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Angiotensin-(1-7) inhibits growth of cardiac myocytes through activation of the mas receptor

Angiotensin II, adhesion, and cardiac fibrosis.

Activation of the ACE2/Ang-(1-7)/Mas pathway reduces oxygen-glucose deprivation-induced tissue swelling, ROS production, and cell death in mouse brain with angiotensin II overproduction.

Nonclassical renin-angiotensin system and renal function.

Role of circulating angiotensin converting enzyme 2 in left ventricular remodeling following myocardial infarction: a prospective controlled study.

ACE2: angiotensin II/angiotensin-(1-7) balance in cardiac and renal injury

Angiotensin (1-7) prevents angiotensin II-induced nociceptive behaviour via inhibition of p38 MAPK phosphorylation mediated through spinal Mas receptors in mice.

Cardiovascular risk reduction with Renin-Angiotensin aldosterone system blockade.

ACE2-Ang (1-7) axis is induced in pressure overloaded rat model.

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1973-1981

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scholarly article

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10.3892/MMR.2017.6848

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2017-06-23T00:00:00Z

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28656296

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5561970

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