The NF-κB1 is a key regulator of acute but not chronic renal injury

about

The NF-κB1 is a key regulator of acute but not chronic renal injury

Item

http://www.wikidata.org/entity/Q38724556

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

The NF-κB1 is a key regulator of acute but not chronic renal injury

@en

type

Item

label

The NF-κB1 is a key regulator of acute but not chronic renal injury

@en

prefLabel

The NF-κB1 is a key regulator of acute but not chronic renal injury

@en

P1476

The NF-κB1 is a key regulator of acute but not chronic renal injury

@en

P2093

Agklinta Kiosia

http://www.w3.org/2001/XMLSchema#string

Amy Fearn

http://www.w3.org/2001/XMLSchema#string

Caroline L Wilson

http://www.w3.org/2001/XMLSchema#string

Christopher Fox

http://www.w3.org/2001/XMLSchema#string

Derek A Mann

http://www.w3.org/2001/XMLSchema#string

Fiona Oakley

http://www.w3.org/2001/XMLSchema#string

Michael G Robson

http://www.w3.org/2001/XMLSchema#string

Neil S Sheerin

http://www.w3.org/2001/XMLSchema#string

Rachel Howarth

http://www.w3.org/2001/XMLSchema#string

P2860

p38 MAPK phosphorylation and NF-kappa B activation in human crescentic glomerulonephritis

NF-κB, the first quarter-century: remarkable progress and outstanding questions

NF-κB in inflammation and renal diseases

NF-kappaB1/p105 regulates lipopolysaccharide-stimulated MAP kinase signaling by governing the stability and function of the Tpl2 kinase

The oncoprotein Bcl-3 directly transactivates through kappa B motifs via association with DNA-binding p50B homodimers

Chronic kidney disease: global dimension and perspectives.

NF-κB1 inhibits TLR-induced IFN-β production in macrophages through TPL-2-dependent ERK activation

C/EBPα and C/EBPα oncoproteins regulate nfkb1 and displace histone deacetylases from NF-κB p50 homodimers to induce NF-κB target genes.

S100A8/A9 (calprotectin) is critical for development of glomerulonephritis and promotes inflammatory leukocyte-renal cell interactions.

Mitogen activated protein kinases as targets for development of novel anti-inflammatory drugs

P304

e2883

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/CDDIS.2017.233

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Anna Moles

Gerhard R Situmorang

P577

2017-06-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28617440

http://www.w3.org/2001/XMLSchema#string

P921

NF-κB

P932

5584573

http://www.w3.org/2001/XMLSchema#string