Glucose Metabolism Reprogrammed by Overexpression of IKKε Promotes Pancreatic Tumor Growth.
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Molecular Drivers of Pancreatic Cancer Pathogenesis: Looking Inward to Move Forward.Resistin potentiates chemoresistance and stemness of breast cancer cells: Implications for racially disparate therapeutic outcomes.Roles for the IKK-Related Kinases TBK1 and IKKε in CancerPGC1β regulates multiple myeloma tumor growth through LDHA-mediated glycolytic metabolismIn vivo genome-wide binding interactions of mouse and human constitutive androstane receptors reveal novel gene targets
P2860
Glucose Metabolism Reprogrammed by Overexpression of IKKε Promotes Pancreatic Tumor Growth.
description
2016 nî lūn-bûn
@nan
2016年の論文
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2016年論文
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2016年論文
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2016年論文
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2016年論文
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2016年論文
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2016年论文
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2016年论文
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2016年论文
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name
Glucose Metabolism Reprogramme ...... motes Pancreatic Tumor Growth.
@en
type
label
Glucose Metabolism Reprogramme ...... motes Pancreatic Tumor Growth.
@en
prefLabel
Glucose Metabolism Reprogramme ...... motes Pancreatic Tumor Growth.
@en
P2093
P2860
P1433
P1476
Glucose Metabolism Reprogramme ...... motes Pancreatic Tumor Growth.
@en
P2093
Aamir Ahmad
Arun Bhardwaj
Girijesh Kumar Patel
Haseeb Zubair
James Elliot Carter
Mohammad Aslam Khan
Sanjeev Kumar Srivastava
Seema Singh
Shafquat Azim
Sumit Arora
P2860
P304
P356
10.1158/0008-5472.CAN-16-1666
P407
P577
2016-10-20T00:00:00Z