Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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P1476

Etiology of alcoholic cardiomyopathy: Mitochondria, oxidative stress and apoptosis.

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P2093

Charles H Lang

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Jennifer L Steiner

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P2860

Crosstalk between Autophagy and Apoptosis: Potential and Emerging Therapeutic Targets for Cardiac Diseases

The good, the bad, and the ugly with alcohol use and abuse on the heart

Long-term alcohol administration inhibits synthesis of both myofibrillar and sarcoplasmic proteins in heart

Alcohol dehydrogenase accentuates ethanol-induced myocardial dysfunction and mitochondrial damage in mice: role of mitochondrial death pathway.

Oxidants, antioxidants and alcohol: implications for skeletal and cardiac muscle.

Chronic ethanol consumption increases myocardial mitochondrial DNA mutations: a potential contribution by mitochondrial topoisomerases

Aldehyde dehydrogenase 2 knockout accentuates ethanol-induced cardiac depression: role of protein phosphatases.

Regulation of mitochondrial biogenesis

Cardiac overexpression of insulin-like growth factor 1 attenuates chronic alcohol intake-induced myocardial contractile dysfunction but not hypertrophy: Roles of Akt, mTOR, GSK3beta, and PTEN.

Alcoholic cardiomyopathy.

P304

125-135

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scholarly article

P356

10.1016/J.BIOCEL.2017.06.009

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P478

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P577

2017-06-09T00:00:00Z

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P698

28606389

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P932

5536333

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