Bactericidal antibiotics promote oxidative damage and programmed cell death in sinonasal epithelial cells.

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Bactericidal antibiotics promote oxidative damage and programmed cell death in sinonasal epithelial cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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Bactericidal antibiotics promo ...... in sinonasal epithelial cells.

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Bactericidal antibiotics promo ...... in sinonasal epithelial cells.

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Bactericidal antibiotics promo ...... in sinonasal epithelial cells.

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P1476

Bactericidal antibiotics promo ...... in sinonasal epithelial cells.

@en

P2093

Anuj Tharakan

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Michael A Kohanski

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Murugappan Ramanathan

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Nyall R London

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P2860

Role of Nrf2 in oxidative stress and toxicity

p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription

Sublethal antibiotic treatment leads to multidrug resistance via radical-induced mutagenesis

A role for mitochondria in NLRP3 inflammasome activation

A common mechanism of cellular death induced by bactericidal antibiotics

Oxidation of the guanine nucleotide pool underlies cell death by bactericidal antibiotics

Bactericidal antibiotics promote reactive oxygen species formation and inflammation in human sinonasal epithelial cells

Mitochondrial formation of reactive oxygen species

Gentamicin rapidly inhibits mitochondrial metabolism in high-frequency cochlear outer hair cells.

Antibiotics induce redox-related physiological alterations as part of their lethality.

P304

359-364

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P31

scholarly article

P356

10.1002/ALR.21914

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Andrew P Lane

P577

2017-01-24T00:00:00Z

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P698

28117948

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P921

antibiotic

P932

5386800

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