Causation by Diesel Exhaust Particles of Endothelial Dysfunctions in Cytotoxicity, Pro-inflammation, Permeability, and Apoptosis Induced by ROS Generation.

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Human peripheral blood mononuclear cells (PBMCs) from smokers release higher levels of IL-1-like cytokines after exposure to combustion-generated ultrafine particles.Transcriptional profiling of human bronchial epithelial cell BEAS-2B exposed to diesel and biomass ultrafine particles.

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Causation by Diesel Exhaust Particles of Endothelial Dysfunctions in Cytotoxicity, Pro-inflammation, Permeability, and Apoptosis Induced by ROS Generation.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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Causation by Diesel Exhaust Pa ...... sis Induced by ROS Generation.

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Causation by Diesel Exhaust Pa ...... sis Induced by ROS Generation.

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Causation by Diesel Exhaust Pa ...... sis Induced by ROS Generation.

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Causation by Diesel Exhaust Pa ...... sis Induced by ROS Generation.

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Chia-Yi Tseng

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Jhih-Syuan Wang

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Ming-Wei Chao

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Diesel exhaust particle-induced alterations of pulmonary phase I and phase II enzymes of rats

Alteration of intracellular cysteine and glutathione levels in alveolar macrophages and lymphocytes by diesel exhaust particle exposure

Involvement of an aldo-keto reductase (AKR1C3) in redox cycling of 9,10-phenanthrenequinone leading to apoptosis in human endothelial cells

Apoptosis: a basic biological phenomenon with wide-ranging implications in tissue kinetics

Diesel exhaust exposure and the risk of lung cancer--a review of the epidemiological evidence

Targeted deficiency or cytosolic truncation of the VE-cadherin gene in mice impairs VEGF-mediated endothelial survival and angiogenesis

Monoclonal antibodies directed to different regions of vascular endothelial cadherin extracellular domain affect adhesion and clustering of the protein and modulate endothelial permeability

Pulmonary oxidative stress, inflammation and cancer: respirable particulate matter, fibrous dusts and ozone as major causes of lung carcinogenesis through reactive oxygen species mechanisms

Persistent endothelial dysfunction in humans after diesel exhaust inhalation

Effects of cell type and culture media on Interleukin-6 secretion in response to environmental particles

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s12012-016-9364-0

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scholarly article

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10.1007/S12012-016-9364-0

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2016-03-10T00:00:00Z

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endothelium

Causation by Diesel Exhaust Particles of Endothelial Dysfunctions in Cytotoxicity, Pro-inflammation, Permeability, and Apoptosis Induced by ROS Generation.

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P2860

P2860

Causation by Diesel Exhaust Particles of Endothelial Dysfunctions in Cytotoxicity, Pro-inflammation, Permeability, and Apoptosis Induced by ROS Generation.

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