about
Dynamic endothelial cell rearrangements drive developmental vessel regressionSensitization of glioblastoma tumor micro-environment to chemo- and immunotherapy by Galectin-1 intranasal knock-down strategyALK1 signaling inhibits angiogenesis by cooperating with the Notch pathway.Netrin-1 controls sympathetic arterial innervation.Thalidomide stimulates vessel maturation and reduces epistaxis in individuals with hereditary hemorrhagic telangiectasia.Identification and functional analysis of endothelial tip cell-enriched genesNon-canonical Wnt signalling modulates the endothelial shear stress flow sensor in vascular remodellingSlit2 signaling through Robo1 and Robo2 is required for retinal neovascularizationSynchronization of endothelial Dll4-Notch dynamics switch blood vessels from branching to expansionThe Robo4 cytoplasmic domain is dispensable for vascular permeability and neovascularization.Boosting the hypoxic response in myeloid cells accelerates resolution of fibrosis and regeneration of the liver in mice.Development of siRNA-loaded chitosan nanoparticles targeting Galectin-1 for the treatment of glioblastoma multiforme via intranasal administration.Development and evaluation of well-tolerated and tumor-penetrating polymeric micelle-based dry powders for inhaled anti-cancer chemotherapy.cAMP-dependent protein kinase A (PKA) regulates angiogenesis by modulating tip cell behavior in a Notch-independent manner.Netrin-1 inhibits sprouting angiogenesis in developing avian embryos.In contrast to agonist monoclonal antibodies, both C-terminal truncated form and full length form of Pleiotrophin failed to activate vertebrate ALK (anaplastic lymphoma kinase)?Correction: dynamic endothelial cell rearrangements drive developmental vessel regression.Semaphorin3A, Neuropilin-1, and PlexinA1 are required for lymphatic valve formation.Glioma-derived galectin-1 regulates innate and adaptive antitumor immunity.Dynamic stroma reorganization drives blood vessel dysmorphia during glioma growth.AMP-activated protein kinase preserves endothelial tight junctions in the coronary microcirculation during sepsis.Development and plasticity of meningeal lymphatic vessels.Angiopoietin-like 4 serum levels on admission for acute myocardial infarction are associated with no-reflow.Connection between cardiac vascular permeability, myocardial edema, and inflammation during sepsis: role of the α1AMP-activated protein kinase isoform.New Folate-Grafted Chitosan Derivative To Improve Delivery of Paclitaxel-Loaded Solid Lipid Nanoparticles for Lung Tumor Therapy by Inhalation.The interaction of heparan sulfate proteoglycans with endothelial transglutaminase-2 limits VEGF165-induced angiogenesis.Protective effects of angiopoietin-like 4 on cerebrovascular and functional damages in ischaemic stroke.Sympathetic Innervation Promotes Arterial Fate by Enhancing Endothelial ERK Activity.Formin-mediated actin polymerization at endothelial junctions is required for vessel lumen formation and stabilization.Impact of video on the understanding and satisfaction of patients receiving informed consent before elective inpatient coronary angiography: A randomized trialAccident vasculaire cérébralRobo4 Maintains Vessel Integrity and Inhibits Angiogenesis by Interacting with UNC5BEndophilin-A2 dependent VEGFR2 endocytosis promotes sprouting angiogenesisMyeloid Derived Suppressor Cells: Key Drivers of Immunosuppression in Ovarian Cancer.Opposite Macrophage Polarization in Different Subsets of Ovarian Cancer: Observation from a Pilot StudyAnatomy and function of the vertebral column lymphatic network in mice
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P50
description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Thomas Mathivet
@ast
Thomas Mathivet
@en
Thomas Mathivet
@es
Thomas Mathivet
@nl
Thomas Mathivet
@sl
type
label
Thomas Mathivet
@ast
Thomas Mathivet
@en
Thomas Mathivet
@es
Thomas Mathivet
@nl
Thomas Mathivet
@sl
prefLabel
Thomas Mathivet
@ast
Thomas Mathivet
@en
Thomas Mathivet
@es
Thomas Mathivet
@nl
Thomas Mathivet
@sl
P106
P1153
6507581379
P21
P31
P496
0000-0001-7761-1684