Exogenous nicotinamide supplementation and moderate physical exercise can attenuate the aging process in skeletal muscle of rats.

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A need for NAD+ in muscle development, homeostasis, and aging.

P2860

Q51149138-D5799123-28DF-4FD9-B229-B5CC5D1CA26F

P2860

Exogenous nicotinamide supplementation and moderate physical exercise can attenuate the aging process in skeletal muscle of rats.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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Exogenous nicotinamide supplem ...... ss in skeletal muscle of rats.

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Exogenous nicotinamide supplem ...... ss in skeletal muscle of rats.

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Exogenous nicotinamide supplem ...... ss in skeletal muscle of rats.

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P1476

Exogenous nicotinamide supplem ...... ss in skeletal muscle of rats.

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P2093

Alexandra Cselko

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Aniko Posa

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Csaba Varga

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Istvan Boldogh

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Margareta Tokodi

http://www.w3.org/2001/XMLSchema#string

Melitta Pajk

http://www.w3.org/2001/XMLSchema#string

Sataro Goto

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P2860

Nicotinamide riboside promotes Sir2 silencing and extends lifespan via Nrk and Urh1/Pnp1/Meu1 pathways to NAD+.

Exercise alters SIRT1, SIRT6, NAD and NAMPT levels in skeletal muscle of aged rats

Short-term caloric restriction, resveratrol, or combined treatment regimens initiated in late-life alter mitochondrial protein expression profiles in a fiber-type specific manner in aged animals

Mitochondrial biogenesis-associated factors underlie the magnitude of response to aerobic endurance training in rats

Age-associated declines in mitochondrial biogenesis and protein quality control factors are minimized by exercise training

SIRT1 is essential for normal cognitive function and synaptic plasticity

Eating habits modulate short term memory and epigenetical regulation of brain derived neurotrophic factor in hippocampus of low- and high running capacity rats.

Combined exercise and insulin-like growth factor-1 supplementation induces neurogenesis in old rats, but do not attenuate age-associated DNA damage

SIRT1 Activity Is Linked to Its Brain Region-Specific Phosphorylation and Is Impaired in Huntington's Disease Mice.

The sirtuin inhibitor nicotinamide enhances neuronal cell survival during acute anoxic injury through AKT, BAD, PARP, and mitochondrial associated "anti-apoptotic" pathways.

P2888

s10522-017-9705-9

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593-600

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scholarly article

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10.1007/S10522-017-9705-9

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Zsolt Radak

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2017-05-05T00:00:00Z

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1085195273

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28477081

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Exogenous nicotinamide supplementation and moderate physical exercise can attenuate the aging process in skeletal muscle of rats.

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P2860

P2860

Exogenous nicotinamide supplementation and moderate physical exercise can attenuate the aging process in skeletal muscle of rats.

description

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