PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

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PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

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PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

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prefLabel

PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

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P1476

PI3K is required for both basal and LPA-induced DNA synthesis in oral carcinoma cells.

@en

P2093

G Hege Thoresen

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Ingvild J Brusevold

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Monica Aasrum

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Paula M De Angelis

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Thoralf Christoffersen

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Vegard Tjomsland

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P2860

Current role of EGF receptor monoclonal antibodies and tyrosine kinase inhibitors in the management of head and neck squamous cell carcinoma

Frequent mutation of the PI3K pathway in head and neck cancer defines predictive biomarkers

Estimates of worldwide burden of cancer in 2008: GLOBOCAN 2008

The emerging role of lysophosphatidic acid in cancer

LPA receptors: subtypes and biological actions

Lysophosphatidic acid 5 receptor induces activation of Na(+)/H(+) exchanger 3 via apical epidermal growth factor receptor in intestinal epithelial cells

Differential requirement of the epidermal growth factor receptor for G protein-mediated activation of transcription factors by lysophosphatidic acid.

Role of LPAR3, PKC and EGFR in LPA-induced cell migration in oral squamous carcinoma cells.

Mechanisms and functions of p38 MAPK signalling.

Adhesive mechanisms regulating invasion and metastasis in oral cancer.

P304

425-432

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P31

scholarly article

P356

10.1111/JOP.12384

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-11-25T00:00:00Z

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P698

26602326

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