All-Trans-Retinoic Acid Stimulates Overexpression of Tumor Protein D52 (TPD52, Isoform 3) and Neuronal Differentiation of IMR-32 Cells.

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All-Trans-Retinoic Acid Stimulates Overexpression of Tumor Protein D52 (TPD52, Isoform 3) and Neuronal Differentiation of IMR-32 Cells.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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All-Trans-Retinoic Acid Stimul ...... fferentiation of IMR-32 Cells.

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All-Trans-Retinoic Acid Stimul ...... fferentiation of IMR-32 Cells.

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All-Trans-Retinoic Acid Stimul ...... fferentiation of IMR-32 Cells.

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P1476

All-Trans-Retinoic Acid Stimul ...... fferentiation of IMR-32 Cells.

@en

P2093

Divya Duscharla

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Manjula Kasula

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Sudha Sravanti Kotapalli

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N-myc is essential during neurogenesis for the rapid expansion of progenitor cell populations and the inhibition of neuronal differentiation

Identification of MAL2, a novel member of the mal proteolipid family, though interactions with TPD52-like proteins in the yeast two-hybrid system

PrLZ, a novel prostate-specific and androgen-responsive gene of the TPD52 family, amplified in chromosome 8q21.1 and overexpressed in human prostate cancer

Tumor protein D52 (TPD52) is overexpressed and a gene amplification target in ovarian cancer

Cyclin D1 is a nuclear protein required for cell cycle progression in G1

Induction of tumorigenesis and metastasis by the murine orthologue of tumor protein D52

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Retinoic acid reduces human neuroblastoma cell migration and invasiveness: effects on DCX, LIS1, neurofilaments-68 and vimentin expression.

P304

4358-4369

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scholarly article

P356

10.1002/JCB.26090

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P433

http://www.w3.org/2001/XMLSchema#string

P478

118

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P50

Chandrashekhar Dasari

Ramesh Ummanni

Karthik Reddy Kami Reddy

P577

2017-04-24T00:00:00Z

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P698

28436114

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