Cellular mechanisms and consequences of glycation in atherosclerosis and obesity. - Wikidata - awgldk - TriplyDB

Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

http://www.wikidata.org/entity/Q38831558

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N-butanol extracts of Morinda citrifolia suppress advanced glycation end products (AGE)-induced inflammatory reactions in endothelial cells through its anti-oxidative properties.2016 ATVB Plenary Lecture: Receptor for Advanced Glycation Endproducts and Implications for the Pathogenesis an Treatment of Cardiometabolic Disorders: Spotlight on the Macrophage.Glycation & the RAGE axis: targeting signal transduction through DIAPH1.TonEBP/NFAT5 haploinsufficiency attenuates hippocampal inflammation in high-fat diet/streptozotocin-induced diabetic mice.Evolution and bad prognostic value of advanced glycation end products after acute heart failure: relation with body composition.From Human Megakaryocytes to Platelets: Effects of Aspirin on High-Mobility Group Box 1/Receptor for Advanced Glycation End Products Axis.Racial differences in circulating levels of the soluble receptor for advanced glycation endproducts in middle-aged and older adults.Responses of Endothelial Cells Towards Ischemic Conditioning Following Acute Myocardial Infarction

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Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

http://www.wikidata.org/entity/Q38831558

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

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Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

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Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

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J.BBADIS.2016.05.005

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Biochimica et Biophysica Acta

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Cellular mechanisms and consequences of glycation in atherosclerosis and obesity.

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Ann Marie Schmidt

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Reduced expression of ATP-binding cassette transporter G1 increases cholesterol accumulation in macrophages of patients with type 2 diabetes mellitus

Interaction of the RAGE cytoplasmic domain with diaphanous-1 is required for ligand-stimulated cellular migration through activation of Rac1 and Cdc42

Tobacco smoke is a source of toxic reactive glycation products

Orally absorbed reactive glycation products (glycotoxins): an environmental risk factor in diabetic nephropathy

Pharmacologic Approaches Against Advanced Glycation End Products (AGEs) in Diabetic Cardiovascular Disease

Oxidative stress and adipocyte biology: focus on the role of AGEs

Signal Transduction in Receptor for Advanced Glycation End Products (RAGE): SOLUTION STRUCTURE OF C-TERMINAL RAGE (ctRAGE) AND ITS BINDING TO mDia1

Glyoxalase I--structure, function and a critical role in the enzymatic defence against glycation

A multimodal RAGE-specific inhibitor reduces amyloid β-mediated brain disorder in a mouse model of Alzheimer disease

Simple non-invasive assessment of advanced glycation endproduct accumulation

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2244-2252

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10.1016/J.BBADIS.2016.05.005

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Alexander Shekhtman

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2016-05-07T00:00:00Z

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atherosclerosis

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5101176

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