Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells.
about
Novel therapeutic mechanisms determine the effectiveness of lipid-core nanocapsules on melanoma models.RNA-binding protein RBM3 prevents NO-induced apoptosis in human neuroblastoma cells by modulating p38 signaling and miR-143Lactobacilli enhance reactive oxygen species-dependent apoptosis-inducing signaling.Enhanced release of primary signals may render intercellular signalling ineffective due to spatial aspects.Special collection: Nitric oxide in cancer.A Mini-Review of Reactive Oxygen Species in Urological Cancer: Correlation with NADPH Oxidases, Angiogenesis, and Apoptosis.Combination of betulinic acid with diazen-1-ium-1,2-diolate nitric oxide moiety donating a novel anticancer candidate.
P2860
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P2860
Increasing the endogenous NO level causes catalase inactivation and reactivation of intercellular apoptosis signaling specifically in tumor cells.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Increasing the endogenous NO l ...... g specifically in tumor cells.
@en
type
label
Increasing the endogenous NO l ...... g specifically in tumor cells.
@en
prefLabel
Increasing the endogenous NO l ...... g specifically in tumor cells.
@en
P2860
P1433
P1476
Increasing the endogenous NO l ...... g specifically in tumor cells.
@en
P2093
Georg Bauer
P2860
P304
P356
10.1016/J.REDOX.2015.07.017
P577
2015-08-24T00:00:00Z