Cigarette smoke augments MUC5AC production via the TLR3-EGFR pathway in airway epithelial cells.
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EGFR Interacts with the Fusion Protein of Respiratory Syncytial Virus Strain 2-20 and Mediates Infection and Mucin ExpressionExtracellular ATP is involved in dsRNA-induced MUC5AC production via P2Y2R in human airway epithelium.NPS2143 Inhibits MUC5AC and Proinflammatory Mediators in Cigarette Smoke Extract (CSE)-Stimulated Human Airway Epithelial Cells.Oxidative stress enhances the expression of IL-33 in human airway epithelial cells.Tobacco Smoke Induces and Alters Immune Responses in the Lung Triggering Inflammation, Allergy, Asthma and Other Lung Diseases: A Mechanistic Review.A Small Molecule BH3-mimetic Suppresses Cigarette Smoke-Induced Mucous Expression in Airway Epithelial Cells
P2860
Cigarette smoke augments MUC5AC production via the TLR3-EGFR pathway in airway epithelial cells.
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name
Cigarette smoke augments MUC5A ...... ay in airway epithelial cells.
@en
type
label
Cigarette smoke augments MUC5A ...... ay in airway epithelial cells.
@en
prefLabel
Cigarette smoke augments MUC5A ...... ay in airway epithelial cells.
@en
P2093
P1476
Cigarette smoke augments MUC5A ...... ay in airway epithelial cells.
@en
P2093
Akira Koarai
Hisatoshi Sugiura
Kazuto Matsunaga
Keiichiro Akamatsu
Kuninobu Kanai
Masakazu Ichinose
Masanori Nakanishi
Takashi Kikuchi
Tomohiro Ichikawa
Tsunahiko Hirano
P304
P356
10.1016/J.RESINV.2015.01.007
P577
2015-03-12T00:00:00Z