RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation. - Wikidata - awgldk - TriplyDB

RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

http://www.wikidata.org/entity/Q38862204

about

Apoptosis and Necroptosis as Host Defense Strategies to Prevent Viral Infection.Programmed Cell Death and Inflammation: Winter Is Coming.RIPped for neuroinflammation.Host response: Neurons loosen the gRIP of death.Down the rabbit hole: Is necroptosis truly an innate response to infection?New advances in CNS immunity against viral infection.Necroptotic cell death in anti-cancer therapy.Recent advances in understanding West Nile virus host immunity and viral pathogenesis.Phenytoin inhibits necroptosis.Intracellular Nucleic Acid Sensing Triggers Necroptosis through Synergistic Type I IFN and TNF Signaling.Caspase-8 deficiency in mouse embryos triggers chronic RIPK1-dependent activation of inflammatory genes, independently of RIPK3.Is Receptor-Interacting Protein Kinase 3 a Viable Therapeutic Target for Mycobacterium tuberculosis Infection?Necroptosis: MLKL Polymerization Overexpression of Chicken IRF7 Increased Viral Replication and Programmed Cell Death to the Avian Influenza Virus Infection Through TGF-Beta/FoxO Signaling Axis in DF-1 Infection with flaviviruses requires BCLXL for cell survival

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RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

http://www.wikidata.org/entity/Q38862204

description

2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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name

RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

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type

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RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

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RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

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J.CELL.2017.03.011

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RIPK3 Restricts Viral Pathogenesis via Cell Death-Independent Neuroinflammation.

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Andrew Oberst

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Annelise G Snyder

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Jennifer Martinez

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Michael Gale

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Tayla M Olsen

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Thomas H Oguin

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Yueh-Ming Loo

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P2860

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

Death in the fast lane: what's next for necroptosis?

IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection

Neuronal CXCL10 directs CD8+ T-cell recruitment and control of West Nile virus encephalitis.

PKR and RNase L Contribute to Protection against Lethal West Nile Virus Infection by Controlling Early Viral Spread in the Periphery and Replication in Neurons

Resistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and Virulence

Caspase 3-Dependent Cell Death of Neurons Contributes to the Pathogenesis of West Nile Virus Encephalitis

A Critical Role for Induced IgM in the Protection against West Nile Virus Infection

Ly6c+ "inflammatory monocytes" are microglial precursors recruited in a pathogenic manner in West Nile virus encephalitis

The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism

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301-313.e11

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scholarly article

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10.1016/J.CELL.2017.03.011

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2

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Brian P Daniels

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2017-03-30T00:00:00Z

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28366204

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