Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process - Wikidata - awgldk - TriplyDB

Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

http://www.wikidata.org/entity/Q38865161

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The Role of Nuclear Antiviral Factors against Invading DNA Viruses: The Immediate Fate of Incoming Viral Genomes Translocation of the papillomavirus L2/vDNA complex across the limiting membrane requires the onset of mitosis Incoming human papillomavirus 16 genome is lost in PML protein-deficient HaCaT keratinocytes.Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus.Human papillomavirus molecular biology.The VPS4 component of the ESCRT machinery plays an essential role in HPV infectious entry and capsid disassembly.The cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry.HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entry.Extracellular conformational changes in the capsid of human papillomaviruses contribute to asynchronous uptake into host cells.Heterotetrameric annexin A2/S100A10 (A2t) is essential for oncogenic human papillomavirus trafficking and capsid disassembly, and protects virions from lysosomal degradation

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Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

http://www.wikidata.org/entity/Q38865161

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

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type

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Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

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Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

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P1433

Journal of Virology

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Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process

@en

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Justyna Broniarczyk

http://www.w3.org/2001/XMLSchema#string

Lawrence Banks

http://www.w3.org/2001/XMLSchema#string

Martina Bergant

http://www.w3.org/2001/XMLSchema#string

Paola Massimi

http://www.w3.org/2001/XMLSchema#string

P2860

Establishment of papillomavirus infection is enhanced by promyelocytic leukemia protein (PML) expression

Endosome maturation

Large scale RNAi reveals the requirement of nuclear envelope breakdown for nuclear import of human papillomaviruses

Entry of human papillomavirus type 16 by actin-dependent, clathrin- and lipid raft-independent endocytosis

Establishment of human papillomavirus infection requires cell cycle progression

Direct binding of retromer to human papillomavirus type 16 minor capsid protein L2 mediates endosome exit during viral infection

Structure, dynamics and functions of promyelocytic leukaemia nuclear bodies

Vesicular trafficking of incoming human papillomavirus 16 to the Golgi apparatus and endoplasmic reticulum requires γ-secretase activity.

Identification of a role for the trans-Golgi network in human papillomavirus 16 pseudovirus infection.

Human papillomavirus types 16, 18, and 31 share similar endocytic requirements for entry.

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8727-8732

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scholarly article

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10.1128/JVI.00722-15

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17

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89

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2015-06-10T00:00:00Z

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26063434

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4524062

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