Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
about
The Role of Nuclear Antiviral Factors against Invading DNA Viruses: The Immediate Fate of Incoming Viral GenomesTranslocation of the papillomavirus L2/vDNA complex across the limiting membrane requires the onset of mitosisIncoming human papillomavirus 16 genome is lost in PML protein-deficient HaCaT keratinocytes.Cruising the cellular highways: How human papillomavirus travels from the surface to the nucleus.Human papillomavirus molecular biology.The VPS4 component of the ESCRT machinery plays an essential role in HPV infectious entry and capsid disassembly.The cellular endosomal protein stannin inhibits intracellular trafficking of human papillomavirus during virus entry.HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entry.Extracellular conformational changes in the capsid of human papillomaviruses contribute to asynchronous uptake into host cells.Heterotetrameric annexin A2/S100A10 (A2t) is essential for oncogenic human papillomavirus trafficking and capsid disassembly, and protects virions from lysosomal degradation
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Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
@en
type
label
Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
@en
prefLabel
Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
@en
P2093
P2860
P356
P1433
P1476
Human Papillomavirus Infectious Entry and Trafficking Is a Rapid Process
@en
P2093
Justyna Broniarczyk
Lawrence Banks
Martina Bergant
Paola Massimi
P2860
P304
P356
10.1128/JVI.00722-15
P407
P577
2015-06-10T00:00:00Z