Aroclor 1254 inhibits cell viability and induces apoptosis of human A549 lung cancer cells by modulating the intracellular Ca(2+) level and ROS production through the mitochondrial pathway.
about
Divergent Effects of Dioxin- or Non-Dioxin-Like Polychlorinated Biphenyls on the Apoptosis of Primary Cell Culture from the Mouse Pituitary Gland.Chlorinated biphenyls effect on estrogen-related receptor expression, steroid secretion, mitochondria ultrastructure but not on mitochondrial membrane potential in Leydig cells.Exposure to decabromodiphenyl ether (BDE-209) produces mitochondrial dysfunction in rat liver and cell death.
P2860
Aroclor 1254 inhibits cell viability and induces apoptosis of human A549 lung cancer cells by modulating the intracellular Ca(2+) level and ROS production through the mitochondrial pathway.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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name
Aroclor 1254 inhibits cell via ...... ugh the mitochondrial pathway.
@en
type
label
Aroclor 1254 inhibits cell via ...... ugh the mitochondrial pathway.
@en
prefLabel
Aroclor 1254 inhibits cell via ...... ugh the mitochondrial pathway.
@en
P2093
P2860
P1476
Aroclor 1254 inhibits cell via ...... ugh the mitochondrial pathway.
@en
P2093
P2860
P304
P356
10.1080/10934529.2015.1019797
P577
2015-01-01T00:00:00Z