miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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MiR-106a-5p inhibits the cell migration and invasion of renal cell carcinoma through targeting PAK5.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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Hongxiong Song

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Hongyi Zhang

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Jixue Gao

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Xiaolong He

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Yadong Ma

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Yayong Qiang

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Yi Li

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Ziming Wang

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Mammary tumorigenesis and metastasis caused by overexpression of insulin receptor substrate 1 (IRS-1) or IRS-2

Cancer statistics, 2013

Oncomirs - microRNAs with a role in cancer

Role of lectin-like oxidized low-density lipoprotein receptor-1 (LOX-1) in the development of hypertensive organ damage

IRS1 is highly expressed in localized breast tumors and regulates the sensitivity of breast cancer cells to chemotherapy, while IRS2 is highly expressed in invasive breast tumors

Divergent roles for IRS-1 and IRS-2 in breast cancer metastasis

MicroRNA-29s could target AKT2 to inhibit gastric cancer cells invasion ability.

Insulin receptor substrates (IRSs) and breast tumorigenesis.

IGF-1R, a target of let-7b, mediates crosstalk between IRS-2/Akt and MAPK pathways to promote proliferation of oral squamous cell carcinoma.

Two new substrates in insulin signaling, IRS5/DOK4 and IRS6/DOK5.

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s13277-015-3605-x

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8389-8398

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scholarly article

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10.1007/S13277-015-3605-X

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2015-05-28T00:00:00Z

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1031069490

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26018509

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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miR-106a* inhibits the proliferation of renal carcinoma cells by targeting IRS-2.

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