ELAS1-mediated inhibition of the cyclin G1-B'γ interaction promotes cancer cell apoptosis via stabilization and activation of p53.

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Comprehensive phenotypic analysis of knockout mice deficient in cyclin G1 and cyclin G2.ELAS1 induces apoptotic death in adenocarcinoma DU145 and squamous-cell carcinoma SAS cancer cells, but not in normal KD cells.

P2860

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ELAS1-mediated inhibition of the cyclin G1-B'γ interaction promotes cancer cell apoptosis via stabilization and activation of p53.

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2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

ELAS1-mediated inhibition of t ...... ization and activation of p53.

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ELAS1-mediated inhibition of t ...... ization and activation of p53.

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ELAS1-mediated inhibition of t ...... ization and activation of p53.

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P1476

ELAS1-mediated inhibition of t ...... ization and activation of p53.

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H Nojima

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S Mukai

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S Ohno

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P2860

Nodal enhances the activity of FoxO3a and its synergistic interaction with Smads to regulate cyclin G2 transcription in ovarian cancer cells

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A truncated isoform of the PP2A B56 subunit promotes cell motility through paxillin phosphorylation

Cyclin G2 is degraded through the ubiquitin-proteasome pathway and mediates the antiproliferative effect of activin receptor-like kinase 7

p53-dependent association between cyclin G and the B' subunit of protein phosphatase 2A

Cyclin G2 associates with protein phosphatase 2A catalytic and regulatory B' subunits in active complexes and induces nuclear aberrations and a G1/S phase cell cycle arrest

The future of peptide-based drugs

Control of cyclin G2 mRNA expression by forkhead transcription factors: novel mechanism for cell cycle control by phosphoinositide 3-kinase and forkhead

CyclinG contributes to G2/M arrest of cells in response to DNA damage

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onc.2015.47

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scholarly article

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10.1038/ONC.2015.47

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ELAS1-mediated inhibition of the cyclin G1-B'γ interaction promotes cancer cell apoptosis via stabilization and activation of p53.

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P2860

P2860

ELAS1-mediated inhibition of the cyclin G1-B'γ interaction promotes cancer cell apoptosis via stabilization and activation of p53.

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