The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
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TRPC6 channel as an emerging determinant of the podocyte injury susceptibility in kidney diseasesTRPC6 May Protect Renal Ischemia-Reperfusion Injury Through Inhibiting Necroptosis of Renal Tubular Epithelial CellsDeletion of TRPC6 Attenuates NMDA Receptor-Mediated Ca2+ Entry and Ca2+-Induced Neurotoxicity Following Cerebral Ischemia and Oxygen-Glucose Deprivation.Podocyte NF-κB is dispensable for the pathogenesis of renal ischemia-reperfusion injury.Association of NF-E2 Related Factor 2 (Nrf2) and inflammatory cytokines in recent onset Type 2 Diabetes Mellitus.
P2860
The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
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2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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2015年學術文章
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name
The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
@en
type
label
The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
@en
prefLabel
The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
@en
P2093
P1476
The role of TRPC6 in oxidative stress-induced podocyte ischemic injury.
@en
P2093
Chang Liao
Jundong Jiao
Kexin Meng
P304
P356
10.1016/J.BBRC.2015.04.054
P407
P577
2015-04-18T00:00:00Z