Combined effects of fine particulate matter and lipopolysaccharide on apoptotic responses in NR8383 macrophages.

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Toll-like receptor 4 in glial inflammatory responses to air pollution in vitro and in vivo.On the translocation of bacteria and their lipopolysaccharides between blood and peripheral locations in chronic, inflammatory diseases: the central roles of LPS and LPS-induced cell death.

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Combined effects of fine particulate matter and lipopolysaccharide on apoptotic responses in NR8383 macrophages.

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Combined effects of fine parti ...... sponses in NR8383 macrophages.

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Combined effects of fine parti ...... sponses in NR8383 macrophages.

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Combined effects of fine parti ...... sponses in NR8383 macrophages.

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Baomiao Ma

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Chaoying Li

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Congyue Xu

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Kai Yue

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Lin Chen

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Lu Liu

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Mingshan Pi

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Qi Xiong

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Qin Ru

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Rihui Wu

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Single-walled carbon nanotubes impair human macrophage engulfment of apoptotic cell corpses

Reactive oxygen species- and nitric oxide-mediated lung inflammation and mitochondrial dysfunction in wild-type and iNOS-deficient mice exposed to diesel exhaust particles

Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases

Bax directly induces release of cytochrome c from isolated mitochondria

Mitochondria and apoptosis

Determinants of the proinflammatory action of ambient particulate matter in immortalized murine macrophages.

Lung toxicity of ambient particulate matter from southeastern U.S. sites with different contributing sources: relationships between composition and effects.

Particulate air pollutants and asthma. A paradigm for the role of oxidative stress in PM-induced adverse health effects.

Health effects associated with exposure to ambient air pollution.

NF-kappaB inhibition is involved in tobacco smoke-induced apoptosis in the lungs of rats

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scholarly article

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10.1080/15287394.2014.993490

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apoptotic process

lipopolysaccharide

Combined effects of fine particulate matter and lipopolysaccharide on apoptotic responses in NR8383 macrophages.

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P2860

Combined effects of fine particulate matter and lipopolysaccharide on apoptotic responses in NR8383 macrophages.

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