Mitochondria and NADPH oxidases are the major sources of TNF-α/cycloheximide-induced oxidative stress in murine intestinal epithelial MODE-K cells.
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Redox Characterization of Functioning Skeletal MuscleMitochondrial energy metabolism disorder and apoptosis: a potential mechanism of postoperative ileusDrp1-dependent mitochondrial fission mediates osteogenic dysfunction in inflammation through elevated production of reactive oxygen species.Placenta Peptide Can Protect Mitochondrial Dysfunction through Inhibiting ROS and TNF-α Generation, by Maintaining Mitochondrial Dynamic Network and by Increasing IL-6 Level during Chronic FatigueThe Protective Roles of ROS-Mediated Mitophagy on 125I Seeds Radiation Induced Cell Death in HCT116 Cells.Differential Effects of CORM-2 and CORM-401 in Murine Intestinal Epithelial MODE-K Cells under Oxidative Stress.Cross talk between increased intracellular zinc (Zn2+) and accumulation of reactive oxygen species in chemical ischemia.Increased mitochondrial ROS generation mediates the loss of the anti-contractile effects of perivascular adipose tissue in high-fat diet obese mice.Activation of Class I histone deacetylases contributes to mitochondrial dysfunction in cardiomyocytes with altered complex activities.
P2860
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P2860
Mitochondria and NADPH oxidases are the major sources of TNF-α/cycloheximide-induced oxidative stress in murine intestinal epithelial MODE-K cells.
description
2015 nî lūn-bûn
@nan
2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年學術文章
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2015年學術文章
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2015年學術文章
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name
Mitochondria and NADPH oxidase ...... tinal epithelial MODE-K cells.
@en
type
label
Mitochondria and NADPH oxidase ...... tinal epithelial MODE-K cells.
@en
prefLabel
Mitochondria and NADPH oxidase ...... tinal epithelial MODE-K cells.
@en
P2093
P1433
P1476
Mitochondria and NADPH oxidase ...... tinal epithelial MODE-K cells.
@en
P2093
Dinesh Babu
Evelien Van Hamme
Georges Leclercq
Romain A Lefebvre
Vera Goossens
P304
P356
10.1016/J.CELLSIG.2015.02.019
P577
2015-02-26T00:00:00Z