The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
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Current understanding of interactions between nanoparticles and the immune systemThe complex cascade of cellular events governing inflammasome activation and IL-1β processing in response to inhaled particlesMolecular mechanisms regulating NLRP3 inflammasome activationThe interplay between inflammation and metabolism in rheumatoid arthritisMyeloid cells in the tumor microenvironment: Role of adenosineEndotoxin contamination: a key element in the interpretation of nanosafety studiesEndocytosis of indium-tin-oxide nanoparticles by macrophages provokes pyroptosis requiring NLRP3-ASC-Caspase1 axis that can be prevented by mesenchymal stem cellsMacrophages participate in local and systemic inflammation induced by amorphous silica nanoparticles through intratracheal instillationATP serves an anti-inflammatory role by enhancing β-defensin-2 response in acute pneumonia of rat.Use of the Microparticle Nanoscale Silicon Dioxide as an Adjuvant To Boost Vaccine Immune Responses against Influenza Virus in Neonatal Mice.Toll-like receptor 9 expression is associated with breast cancer sensitivity to the growth inhibitory effects of bisphosphonates in vitro and in vivo.Effect of TiO₂ Nanoparticles on Inflammasome-Mediated Airway Inflammation and Responsiveness.Human Scavenger Receptor A1-Mediated Inflammatory Response to Silica Particle Exposure Is Size Specific.Purinergic receptors: new targets for the treatment of gout and fibrosis.Purinergic signaling during intestinal inflammation.Why the Immune System Should Be Concerned by Nanomaterials?Surface Coating of Nanoparticles Reduces Background Inflammatory Activity while Increasing Particle Uptake and DeliverySilver Nanoparticle-Induced Autophagic-Lysosomal Disruption and NLRP3-Inflammasome Activation in HepG2 Cells Is Size-Dependent.Mechanistic insight into the impact of nanomaterials on asthma and allergic airway disease.In Search of a Converging Cellular Mechanism in Nanotoxicology and Nanomedicine in the Treatment of Cancer.Silica nanoparticles induce NLRP3 inflammasome activation in human primary immune cells.Anti-inflammatory Nanomedicine for Cardiovascular Disease.Connexins in Cardiovascular and Neurovascular Health and Disease: Pharmacological Implications.Differential proteomics highlights macrophage-specific responses to amorphous silica nanoparticles.Polymeric Nanoparticles Induce NLRP3 Inflammasome Activation and Promote Breast Cancer Metastasis.In Vitro and In Vivo Short-Term Pulmonary Toxicity of Differently Sized Colloidal Amorphous SiO₂.Modulation of Immune Responses by Particulate Materials.ATP-induced IL-1β secretion is selectively impaired in microglia as compared to hematopoietic macrophages.Purinergic Regulation of Neutrophil Function.Determining the relationship between nanoparticle characteristics and immunotoxicity: key challenges and approaches.Dual Effect of Hepatic Macrophages on Liver Ischemia and Reperfusion Injury during Liver Transplantation.The IL-33 Receptor ST2 Regulates Pulmonary Inflammation and Fibrosis to Bleomycin.P2Y2R-mediated inflammasome activation is involved in tumor progression in breast cancer cells and in radiotherapy-resistant breast cancer
P2860
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P2860
The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
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2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
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2015年學術文章
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name
The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
@en
type
label
The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
@en
prefLabel
The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
@en
P2093
P2860
P356
P1476
The NLRP3 inflammasome is activated by nanoparticles through ATP, ADP and adenosine.
@en
P2093
A Gombault
B Villeret
F Rassendren
I Couillin
P2860
P356
10.1038/CDDIS.2014.576
P577
2015-02-05T00:00:00Z