CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

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CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

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CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

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CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

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P1476

CCL11 promotes angiogenic activity by activating the PI3K/Akt pathway in HUVECs.

@en

P2093

Byung Pil Cho

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Byung Young Choi

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Won Gil Cho

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Yeo Wool Kang

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Young Chul Yang

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P2860

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Cloning of the human eosinophil chemoattractant, eotaxin. Expression, receptor binding, and functional properties suggest a mechanism for the selective recruitment of eosinophils

Angiogenesis in cancer and other diseases

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The HMG-CoA reductase inhibitor simvastatin activates the protein kinase Akt and promotes angiogenesis in normocholesterolemic animals.

Eotaxin and eosinophil recruitment: implications for human disease.

Endothelial cell metabolism: parallels and divergences with cancer cell metabolism

Endothelial cell migration during angiogenesis.

Role of Akt signaling in vascular homeostasis and angiogenesis.

P304

416-421

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P31

scholarly article

P356

10.1080/10799893.2017.1298132

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Jun Young Park

P577

2017-03-09T00:00:00Z

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P698

28279120

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