Transforming growth factor-β promotes prostate bone metastasis through induction of microRNA-96 and activation of the mTOR pathway.
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Dysregulation and functional roles of miR-183-96-182 cluster in cancer cell proliferation, invasion and metastasisKrüppel-like factor 4 (KLF4) regulates the miR-183~96~182 cluster under physiologic and pathologic conditions.miR-3188 regulates nasopharyngeal carcinoma proliferation and chemosensitivity through a FOXO1-modulated positive feedback loop with mTOR-p-PI3K/AKT-c-JUN.Targeting oncomiRNAs and mimicking tumor suppressor miRNAs: Νew trends in the development of miRNA therapeutic strategies in oncology (Review).EGF Receptor Promotes Prostate Cancer Bone Metastasis by Downregulating miR-1 and Activating TWIST1.Androgen receptor regulates SRC expression through microRNA-203KLF4 functions as an activator of the androgen receptor through reciprocal feedback.TGF-β1 promotes colorectal cancer immune escape by elevating B7-H3 and B7-H4 via the miR-155/miR-143 axisMiRNA 34a: a therapeutic target for castration-resistant prostate cancer.Dissecting the role of microRNAs in prostate cancer metastasis: implications for the design of novel therapeutic approaches.Deciphering the function of non-coding RNAs in prostate cancer.MicroRNA Exert Macro Effects on Cancer Bone Metastasis.TCF7 is suppressed by the androgen receptor via microRNA-1-mediated downregulation and is involved in the development of resistance to androgen deprivation in prostate cancer.Emerging Role of MicroRNAs in mTOR Signaling.Epigenetic and oncogenic regulation of SLC16A7 (MCT2) results in protein over-expression, impacting on signalling and cellular phenotypes in prostate cancerOncogenic miR-210-3p promotes prostate cancer cell EMT and bone metastasis via NF-κB signaling pathway.Loss of miR-449a-caused PrLZ overexpression promotes prostate cancer metastasisPRAS40 signaling in tumor.Molecular determinants of prostate cancer metastasis.Downregulation of miR-141-3p promotes bone metastasis via activating NF-κB signaling in prostate cancer.ITGB1-dependent upregulation of Caveolin-1 switches TGFβ signalling from tumour-suppressive to oncogenic in prostate cancer.Downregulation of miR‑19a‑3p promotes invasion, migration and bone metastasis via activating TGF‑β signaling in prostate cancer.Disruption of ETV6 leads to TWIST1-dependent progression and resistance to epidermal growth factor receptor tyrosine kinase inhibitors in prostate cancer.Cell-autonomous and cell non-autonomous downregulation of tumor suppressor DAB2IP by microRNA-149-3p promotes aggressiveness of cancer cells.mTOR signaling-related MicroRNAs and Cancer involvement.Transcriptional downregulation of miR-133b by REST promotes prostate cancer metastasis to bone via activating TGF-β signaling.MicroRNAs in prostate cancer: From function to biomarker discoveryPromotes Schistosomiasis Hepatic Fibrosis in Mice by Suppressing
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P2860
Transforming growth factor-β promotes prostate bone metastasis through induction of microRNA-96 and activation of the mTOR pathway.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
2014年论文
@zh
2014年论文
@zh-cn
name
Transforming growth factor-β p ...... ctivation of the mTOR pathway.
@en
type
label
Transforming growth factor-β p ...... ctivation of the mTOR pathway.
@en
prefLabel
Transforming growth factor-β p ...... ctivation of the mTOR pathway.
@en
P2093
P2860
P356
P1433
P1476
Transforming growth factor-β p ...... ctivation of the mTOR pathway.
@en
P2093
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P2888
P304
P356
10.1038/ONC.2014.414
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P577
2014-12-22T00:00:00Z
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P6179
1018853669