The ER Stress Sensor PERK Coordinates ER-Plasma Membrane Contact Site Formation through Interaction with Filamin-A and F-Actin Remodeling.

The ER Stress Sensor PERK Coordinates ER-Plasma Membrane Contact Site Formation through Interaction with Filamin-A and F-Actin Remodeling.

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http://www.wikidata.org/entity/Q38941393
C. elegans SUP-46, an HNRNPM family RNA-binding protein that prevents paternally-mediated epigenetic sterility.Cancer: Untethering Mitochondria from the Endoplasmic Reticulum?IP3 Receptor-Mediated Calcium Signaling and Its Role in Autophagy in Cancer.Mitochondria-Associated Membranes As Networking Platforms and Regulators of Cancer Cell Fate.Membrane dynamics and organelle biogenesis-lipid pipelines and vesicular carriers.Overexpression of sphingosine-1-phosphate lyase protects insulin-secreting cells against cytokine toxicity.Contribution of the cytoskeleton to mechanosensitivity reported by dinoflagellate bioluminescence.ER-driven membrane contact sites: Evolutionary conserved machineries for stress response and autophagy regulation?PERK interacts with FLNA to regulate ER-PM contact sites.Cell death and immunity in cancer: From danger signals to mimicry of pathogen defense responses.PERK and filamin A in actin cytoskeleton remodeling at ER-plasma membrane contact sites.Cortical actin contributes to spatial organization of ER-PM junctions.SAC1 degrades its lipid substrate PtdIns4P in the endoplasmic reticulum to maintain a steep chemical gradient with donor membranes.The protein kinase PERK/EIF2AK3 regulates proinsulin processing not via protein synthesis but by controlling endoplasmic reticulum chaperones.Inhibition of Sec61-dependent translocation by mycolactone uncouples the integrated stress response from ER stress, driving cytotoxicity via translational activation of ATF4.Role of Mitochondria-Associated ER Membranes in Calcium Regulation in Cancer-Specific Settings.Ceramide Metabolism Balance, a Multifaceted Factor in Critical Steps of Breast Cancer Development
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The ER Stress Sensor PERK Coordinates ER-Plasma Membrane Contact Site Formation through Interaction with Filamin-A and F-Actin Remodeling.

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http://www.wikidata.org/entity/Q38941393
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年学术文章
@wuu
2017年学术文章
@zh-cn
2017年学术文章
@zh-hans
2017年学术文章
@zh-my
2017年学术文章
@zh-sg
2017年學術文章
@yue
2017年學術文章
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2017年學術文章
@zh-hant
name
The ER Stress Sensor PERK Coor ...... amin-A and F-Actin Remodeling.
@en
type
label
The ER Stress Sensor PERK Coor ...... amin-A and F-Actin Remodeling.
@en
prefLabel
The ER Stress Sensor PERK Coor ...... amin-A and F-Actin Remodeling.
@en
P1433
P1476
P2093
P304
P31
P356
P433
P478
P50
P577
P698
P356
P1433
P1476
The ER Stress Sensor PERK Coor ...... lamin-A and F-Actin Remodeling
@en
P2093
P304
P31
P356
10.1016/J.MOLCEL.2017.01.020
P433
P478
P50
P577
P698