Growth hormone enhances LNCaP prostate cancer cell motility.

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Minireview: Were the IGF Signaling Inhibitors All Bad?Androgen Receptor Regulation of Local Growth Hormone in Prostate Cancer Cells.

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Growth hormone enhances LNCaP prostate cancer cell motility.

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2014 nî lūn-bûn

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2014年の論文

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2014年学术文章

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2014年学术文章

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2014年学术文章

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2014年学术文章

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Growth hormone enhances LNCaP prostate cancer cell motility.

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Growth hormone enhances LNCaP prostate cancer cell motility.

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Growth hormone enhances LNCaP prostate cancer cell motility.

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P1476

Growth hormone enhances LNCaP prostate cancer cell motility.

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P2093

Alona O Nakonechnaya

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Brian M Shewchuk

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P2860

Rho GTPases in cell biology

Growth hormone stimulates the tyrosine phosphorylation and association of p125 focal adhesion kinase (FAK) with JAK2. Fak is not required for stat-mediated transcription

Evidence that insulin-like growth factor I and growth hormone are required for prostate gland development

Mammalian Rho GTPases: new insights into their functions from in vivo studies

Reduced cell motility and enhanced focal adhesion contact formation in cells from FAK-deficient mice

The role of cell motility in prostate cancer.

Transcription factor Stat3 stimulates metastatic behavior of human prostate cancer cells in vivo, whereas Stat5b has a preferential role in the promotion of prostate cancer cell viability and tumor growth.

Proliferation and invasion: plasticity in tumor cells.

Morphogenic and tumorigenic potentials of the mammary growth hormone/growth hormone receptor system.

Role of integrins in cell invasion and migration.

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97-105

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scholarly article

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10.3109/07435800.2014.966383

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2014-10-20T00:00:00Z

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267101666

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25330355

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Growth hormone enhances LNCaP prostate cancer cell motility.

about

P2860

P2860

Growth hormone enhances LNCaP prostate cancer cell motility.

description

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type

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P1433

P1476

P2093

P2860

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P1476

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P2860

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