Interleukin-6 induces neuroendocrine differentiation (NED) through suppression of RE-1 silencing transcription factor (REST). - Wikidata - awgldk - TriplyDB

Interleukin-6 induces neuroendocrine differentiation (NED) through suppression of RE-1 silencing transcription factor (REST).

Interleukin-6 induces neuroendocrine differentiation (NED) through suppression of RE-1 silencing transcription factor (REST).

http://www.wikidata.org/entity/Q38995864

about

Volatile Organic Compound Gamma-Butyrolactone Released upon Herpes Simplex Virus Type -1 Acute Infection Modulated Membrane Potential and Repressed Viral Infection in Human Neuron-Like Cells MAOA-a novel decision maker of apoptosis and autophagy in hormone refractory neuroendocrine prostate cancer cells.Antiandrogens Inhibit ABCB1 Efflux and ATPase Activity and Reverse Docetaxel Resistance in Advanced Prostate Cancer Influences of laparoscopic-assisted gastrectomy and open gastrectomy on serum interleukin-6 levels in patients with gastric cancer among Asian populations: a systematic review Up-Regulated Expression of LAMP2 and Autophagy Activity during Neuroendocrine Differentiation of Prostate Cancer LNCaP Cells REST is a crucial regulator for acquiring EMT-like and stemness phenotypes in hormone-refractory prostate cancer.c-MYC drives histone demethylase PHF8 during neuroendocrine differentiation and in castration-resistant prostate cancer.The cannabinoid WIN 55,212-2 prevents neuroendocrine differentiation of LNCaP prostate cancer cells.Niclosamide suppresses cell migration and invasion in enzalutamide resistant prostate cancer cells via Stat3-AR axis inhibition.Elevated circulating tissue inhibitor of metalloproteinase 1 (TIMP-1) levels are associated with neuroendocrine differentiation in castration resistant prostate cancer.Emerging trends in the evaluation and management of small cell prostate cancer: a clinical and molecular perspective.Prostate-specific IL-6 transgene autonomously induce prostate neoplasm through amplifying inflammation in the prostate and peri-prostatic adipose tissue.Establishment of a neuroendocrine prostate cancer model driven by the RNA splicing factor SRRM4.Induction of Neuroendocrine Differentiation in Prostate Cancer Cells by Dovitinib (TKI-258) and its Therapeutic Implications.SRRM4 Expression and the Loss of REST Activity May Promote the Emergence of the Neuroendocrine Phenotype in Castration-Resistant Prostate Cancer.Alternative RNA splicing of the MEAF6 gene facilitates neuroendocrine prostate cancer progression.Implications of PI3K/AKT inhibition on REST protein stability and neuroendocrine phenotype acquisition in prostate cancer cells.REST Final-Exon-Truncating Mutations Cause Hereditary Gingival Fibromatosis.Effect of A549 neuroendocrine differentiation on cytotoxic immune response.Development of Neuroendocrine Prostate Cancers by the Ser/Arg Repetitive Matrix 4-Mediated RNA Splicing Network.Roles of Alternative RNA Splicing of the Bif-1 Gene by SRRM4 During the Development of Treatment-induced Neuroendocrine Prostate Cancer.

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P2860

Interleukin-6 induces neuroendocrine differentiation (NED) through suppression of RE-1 silencing transcription factor (REST).

http://www.wikidata.org/entity/Q38995864

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

@zh-cn

name

Interleukin-6 induces neuroend ...... g transcription factor (REST).

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Interleukin-6 induces neuroend ...... g transcription factor (REST).

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Interleukin-6 induces neuroend ...... g transcription factor (REST).

@en

P2093

Allen C Gao

http://www.w3.org/2001/XMLSchema#string

Chengfei Liu

http://www.w3.org/2001/XMLSchema#string

Nagalakshmi Nadiminty

http://www.w3.org/2001/XMLSchema#string

Wei Lou

http://www.w3.org/2001/XMLSchema#string

Yezi Zhu

http://www.w3.org/2001/XMLSchema#string

Yuanyuan Cui

http://www.w3.org/2001/XMLSchema#string

P2860

SCFbeta-TRCP controls oncogenic transformation and neural differentiation through REST degradation

Interleukin-6- and cyclic AMP-mediated signaling potentiates neuroendocrine differentiation of LNCaP prostate tumor cells.

A genetic screen for candidate tumor suppressors identifies REST

REST-VP16 activates multiple neuronal differentiation genes in human NT2 cells

REST in good times and bad: roles in tumor suppressor and oncogenic activities.

LNCaP prostate cancer cells with autocrine interleukin-6 expression are resistant to IL-6-induced neuroendocrine differentiation due to increased expression of suppressors of cytokine signaling

Neuroendocrine-like prostate cancer cells: neuroendocrine transdifferentiation of prostate adenocarcinoma cells.

RE-1-silencing transcription factor shows tumor-suppressor functions and negatively regulates the oncogenic TAC1 in breast cancer cells.

Neuroendocrine differentiation in the progression of prostate cancer.

REST mediates androgen receptor actions on gene repression and predicts early recurrence of prostate cancer.

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1086-1094

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scholarly article

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10.1002/PROS.22819

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11

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74

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2014-05-12T00:00:00Z

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262232516

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24819501

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