Metabolism of arsenic trioxide in acute promyelocytic leukemia cells.
about
Oncogenomic disruptions in arsenic-induced carcinogenesis.Methylated arsenic metabolites bind to PML protein but do not induce cellular differentiation and PML-RARα protein degradationDeficiency of SUMO-specific protease 1 induces arsenic trioxide-mediated apoptosis by regulating XBP1 activity in human acute promyelocytic leukemia.Arsenic (+3 oxidation state) methyltransferase is a specific but replaceable factor against arsenic toxicity.Redistribution of cell cycle by arsenic trioxide is associated with demethylation and expression changes of cell cycle related genes in acute promyelocytic leukemia cell line (NB4).
P2860
Metabolism of arsenic trioxide in acute promyelocytic leukemia cells.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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2014年论文
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name
Metabolism of arsenic trioxide in acute promyelocytic leukemia cells.
@en
type
label
Metabolism of arsenic trioxide in acute promyelocytic leukemia cells.
@en
prefLabel
Metabolism of arsenic trioxide in acute promyelocytic leukemia cells.
@en
P2093
P2860
P356
P1476
Metabolism of arsenic trioxide in acute promyelocytic leukemia cells
@en
P2093
Ali Khaleghian
Ardeshir Ghavamzadeh
Kamran Alimoghaddam
Shahin Ahmadian
P2860
P304
P356
10.1002/JCB.24838
P50
P577
2014-10-01T00:00:00Z