Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
about
PD-1, PD-L1 and PD-L2 Gene Expression on T-Cells and Natural Killer Cells Declines in Conjunction with a Reduction in PD-1 Protein during the Intensive Phase of Tuberculosis TreatmentPPD-induced monocyte mitochondrial damage is associated with a protective effect to develop tuberculosis in BCG vaccinated individuals: A cohort study.Approaching a diagnostic point-of-care test for pediatric tuberculosis through evaluation of immune biomarkers across the clinical disease spectrumMolecular Signatures of Immunity and Immunogenicity in Infection and Vaccination.Influence of the polymorphism of the DUSP14 gene on the expression of immune-related genes and development of pulmonary tuberculosis.
P2860
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
description
2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@en
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@nl
type
label
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@en
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@nl
prefLabel
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@en
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@nl
P2093
P2860
P356
P1476
Dysregulation of Apoptosis Is a Risk Factor for Tuberculosis Disease Progression.
@en
P2093
Jeroen Maertzdorf
Marielle C Haks
Olumuyiwa Owolabi
Philip C Hill
Simon Donkor
Stefan H E Kaufmann
Thomas O J P Elliott
Tom H M Ottenhoff
P2860
P304
P356
10.1093/INFDIS/JIV238
P407
P577
2015-04-20T00:00:00Z