Vessel pruning or healing: endothelial metabolism as a novel target? - Wikidata - awgldk - TriplyDB

Vessel pruning or healing: endothelial metabolism as a novel target?

Vessel pruning or healing: endothelial metabolism as a novel target?

http://www.wikidata.org/entity/Q39024164

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The Role of Tumor Microenvironment in Chemoresistance: To Survive, Keep Your Enemies Closer.Synergies of Targeting Tumor Angiogenesis and Immune Checkpoints in Non-Small Cell Lung Cancer and Renal Cell Cancer: From Basic Concepts to Clinical Reality.Collateral Damage Intended-Cancer-Associated Fibroblasts and Vasculature Are Potential Targets in Cancer Therapy.Suggested Mechanisms of Tracheal Occlusion Mediated Accelerated Fetal Lung Growth: A Case for Heterogeneous Topological Zones.Tumor vessel disintegration by maximum tolerable PFKFB3 blockade.Angiogenic Factors produced by Hypoxic Cells are a leading driver of Anastomoses in Sprouting Angiogenesis-a computational study.The Warburg Effect in Endothelial Cells and its Potential as an Anti-angiogenic Target in Cancer

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P2860

Vessel pruning or healing: endothelial metabolism as a novel target?

http://www.wikidata.org/entity/Q39024164

description

2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

Vessel pruning or healing: endothelial metabolism as a novel target?

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Vessel pruning or healing: endothelial metabolism as a novel target?

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type

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Vessel pruning or healing: endothelial metabolism as a novel target?

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Vessel pruning or healing: endothelial metabolism as a novel target?

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Vessel pruning or healing: endothelial metabolism as a novel target?

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Vessel pruning or healing: endothelial metabolism as a novel target?

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Expert Opinion on Therapeutic Targets

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Vessel pruning or healing: endothelial metabolism as a novel target?

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Andreas Pircher

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Anna Rita Cantelmo

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Joanna Kalucka

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Creative Commons Attribution 4.0 International

P2860

Double antiangiogenic protein, DAAP, targeting VEGF-A and angiopoietins in tumor angiogenesis, metastasis, and vascular leakage

HRG inhibits tumor growth and metastasis by inducing macrophage polarization and vessel normalization through downregulation of PlGF

Modes of resistance to anti-angiogenic therapy

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

Contrasting actions of selective inhibitors of angiopoietin-1 and angiopoietin-2 on the normalization of tumor blood vessels

Tumor refractoriness to anti-VEGF therapy

VEGF pathway targeting agents, vessel normalization and tumor drug uptake: from bench to bedside

Inhibition of Immune Checkpoints and Vascular Endothelial Growth Factor as Combination Therapy for Metastatic Melanoma: An Overview of Rationale, Preclinical Evidence, and Initial Clinical Data

Ways to enhance lymphocyte trafficking into tumors and fitness of tumor infiltrating lymphocytes

Resistance and escape from antiangiogenesis therapy: clinical implications and future strategies

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Peter Carmeliet

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