Reno-Cerebral Reflex Activates the Renin-Angiotensin System, Promoting Oxidative Stress and Renal Damage After Ischemia-Reperfusion Injury.

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Sympathetic Nerve Hyperactivity in the Spleen: Causal for Nonpathogenic-Driven Chronic Immune-Mediated Inflammatory Diseases (IMIDs)?Risk of Incident Non-Valvular Atrial Fibrillation after Dialysis-Requiring Acute Kidney Injury

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P2860

Reno-Cerebral Reflex Activates the Renin-Angiotensin System, Promoting Oxidative Stress and Renal Damage After Ischemia-Reperfusion Injury.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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type

Item

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Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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P1476

Reno-Cerebral Reflex Activates ...... r Ischemia-Reperfusion Injury.

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P2093

Aiqing Li

http://www.w3.org/2001/XMLSchema#string

Christopher S Wilcox

http://www.w3.org/2001/XMLSchema#string

Chunyi Wu

http://www.w3.org/2001/XMLSchema#string

Fan Fan Hou

http://www.w3.org/2001/XMLSchema#string

Jiawen Li

http://www.w3.org/2001/XMLSchema#string

Shuang Cui

http://www.w3.org/2001/XMLSchema#string

Youhua Liu

http://www.w3.org/2001/XMLSchema#string

Zhanmei Zhou

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P2860

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Effects of the antioxidant drug tempol on renal oxygenation in mice with reduced renal mass.

The NADPH oxidase subunit NOX4 is a new target gene of the hypoxia-inducible factor-1.

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415-432

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scholarly article

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10.1089/ARS.2016.6827

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2016-12-28T00:00:00Z

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28030955

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reperfusion injury

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5549812

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Reno-Cerebral Reflex Activates the Renin-Angiotensin System, Promoting Oxidative Stress and Renal Damage After Ischemia-Reperfusion Injury.

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P2860

P2860

Reno-Cerebral Reflex Activates the Renin-Angiotensin System, Promoting Oxidative Stress and Renal Damage After Ischemia-Reperfusion Injury.

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