Propofol limits microglial activation after experimental brain trauma through inhibition of nicotinamide adenine dinucleotide phosphate oxidase.
about
Alzheimer’s disease and chronic traumatic encephalopathy: distinct but possibly overlapping disease entitiesThe choice of general anesthetics may not affect neuroinflammation and impairment of learning and memory after surgery in elderly rats.Protective effect of propofol preconditioning on ischemia-reperfusion injury in human hepatocyte.Comparison of neurodegeneration and cognitive impairment in neonatal mice exposed to propofol or isoflurane.Repeated mild traumatic brain injury causes chronic neuroinflammation, changes in hippocampal synaptic plasticity, and associated cognitive deficits.Chronic decrease in wakefulness and disruption of sleep-wake behavior after experimental traumatic brain injury.Propofol Attenuates Small Intestinal Ischemia Reperfusion Injury through Inhibiting NADPH Oxidase Mediated Mast Cell ActivationPropofol ameliorates calpain-induced collapsin response mediator protein-2 proteolysis in traumatic brain injury in ratsWhite matter damage after traumatic brain injury: A role for damage associated molecular patterns.Neuronal loss as evidenced by automated quantification of neuronal density following moderate and severe traumatic brain injury in rats.Propofol Protects Against H2O2-Induced Oxidative Injury in Differentiated PC12 Cells via Inhibition of Ca(2+)-Dependent NADPH Oxidase.Propofol Affects Neurodegeneration and Neurogenesis by Regulation of Autophagy via Effects on Intracellular Calcium Homeostasis.Histamine H3 Receptor Antagonist Prevents Memory Deficits and Synaptic Plasticity Disruption Following Isoflurane Exposure.Protective Role of Apocynin via Suppression of Neuronal Autophagy and TLR4/NF-κB Signaling Pathway in a Rat Model of Traumatic Brain Injury.Propofol attenuates BV2 microglia inflammation via NMDA receptor inhibition.Propofol Attenuates Inflammatory Response in LPS-Activated Microglia by Regulating the miR-155/SOCS1 Pathway.NADPH oxidases in traumatic brain injury - Promising therapeutic targets?A single primary blast-induced traumatic brain injury in rodent model causes cell-type dependent increase in NADPH oxidase isoforms in vulnerable brain regions.Astragalus polysaccharide attenuates lipopolysaccharide-induced inflammatory responses in microglial cells: regulation of protein kinase B and nuclear factor-κB signaling.Delayed treatment of propofol inhibits lipopolysaccharide-induced inflammation in microglia through the PI3K/PKB pathway.Propofol Protects Rat Cardiomyocytes from Anthracycline-Induced Apoptosis by Regulating MicroRNA-181a In Vitro and In Vivo.
P2860
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P2860
Propofol limits microglial activation after experimental brain trauma through inhibition of nicotinamide adenine dinucleotide phosphate oxidase.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@en
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@nl
type
label
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@en
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@nl
prefLabel
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@en
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@nl
P2093
P1433
P1476
Propofol limits microglial act ...... inucleotide phosphate oxidase.
@en
P2093
Alan I Faden
Boris Sabirzhanov
Charles Jeremy Bengson
Juliane Faden
Junfang Wu
Katherine Cardiff
Kelsey Guanciale
Marie Hanscom
Shruti V Kabadi
P304
P356
10.1097/ALN.0000000000000020
P407
P577
2013-12-01T00:00:00Z