Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass. - Wikidata - awgldk - TriplyDB

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

http://www.wikidata.org/entity/Q39099310

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Myostatin: expanding horizons The quasi-parallel lives of satellite cells and atrophying muscle Mechanisms of muscle wasting in chronic kidney disease STAT3 signaling controls satellite cell expansion and skeletal muscle repair.Low-frequency electrical stimulation attenuates muscle atrophy in CKD--a potential treatment strategy Inhibition of Stat3 activation suppresses caspase-3 and the ubiquitin-proteasome system, leading to preservation of muscle mass in cancer cachexia Sunitinib prevents cachexia and prolongs survival of mice bearing renal cancer by restraining STAT3 and MuRF-1 activation in muscle.Acupuncture plus Low-Frequency Electrical Stimulation (Acu-LFES) Attenuates Diabetic Myopathy by Enhancing Muscle Regeneration.Urea and Ammonia Metabolism and the Control of Renal Nitrogen Excretion.Diets for patients with chronic kidney disease, should we reconsider?Small-molecule targeting of signal transducer and activator of transcription (STAT) 3 to treat non-small cell lung cancer.STAT-3 contributes to pulmonary fibrosis through epithelial injury and fibroblast-myofibroblast differentiation.Lewis lung carcinoma regulation of mechanical stretch-induced protein synthesis in cultured myotubes.CKD Stimulates Muscle Protein Loss Via Rho-associated Protein Kinase 1 Activation.Inhibition of myostatin in mice improves insulin sensitivity via irisin-mediated cross talk between muscle and adipose tissues.Signal transducer and activator of transcription 3 signaling as a potential target to treat muscle wasting diseases.STAT3 in the systemic inflammation of cancer cachexia.Janus kinase inhibition prevents cancer- and myocardial infarction-mediated diaphragm muscle weakness in mice.Small-molecule inhibition of STAT3 in radioresistant head and neck squamous cell carcinoma Differential RNA Expression Profile of Skeletal Muscle Induced by Experimental Autoimmune Myasthenia Gravis in Rats.STAT3 Undergoes Acetylation-dependent Mitochondrial Translocation to Regulate Pyruvate Metabolism.Inhibition of Stat3 signaling ameliorates atrophy of the soleus muscles in mice lacking the vitamin D receptor.Suppression of muscle wasting by the plant-derived compound ursolic acid in a model of chronic kidney disease.Muscle wasting in end-stage renal disease promulgates premature death: established, emerging and potential novel treatment strategies.Acupuncture plus low-frequency electrical stimulation (Acu-LFES) attenuates denervation-induced muscle atrophy.Differential control of muscle mass in type 1 and type 2 diabetes mellitus.Sarcopenia, obesity, and mortality in US adults with and without chronic kidney disease.Overload-mediated skeletal muscle hypertrophy is not impaired by loss of myofiber STAT3.Muscle atrophy in patients with Type 2 Diabetes Mellitus: roles of inflammatory pathways, physical activity and exercise.Modulation of Myostatin/Hepatocyte Growth Factor Balance by Different Hemodialysis Modalities.The ubiquitin proteasome system in atrophying skeletal muscle: roles and regulation.Pathogenic role of anti-signal recognition protein and anti-3-Hydroxy-3-methylglutaryl-CoA reductase antibodies in necrotizing myopathies: Myofiber atrophy and impairment of muscle regeneration in necrotizing autoimmune myopathies.Skeletal muscle inflammation and atrophy in heart failure.Parathyroid hormone stimulates adipose tissue browning: a pathway to muscle wasting.Indoxyl sulfate potentiates skeletal muscle atrophy by inducing the oxidative stress-mediated expression of myostatin and atrogin-1.Activation of the SDF1/CXCR4 pathway retards muscle atrophy during cancer cachexia.The Effect of Resistance Exercise on Inflammatory and Myogenic Markers in Patients with Chronic Kidney Disease.MicroRNA-23a and MicroRNA-27a Mimic Exercise by Ameliorating CKD-Induced Muscle Atrophy.STAT3 inhibition prevents lung inflammation, remodeling, and accumulation of Th2 and Th17 cells in a murine asthma model.Serum Glucocorticoid-Regulated Kinase 1 Blocks CKD-Induced Muscle Wasting Via Inactivation of FoxO3a and Smad2/3.

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P2860

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

http://www.wikidata.org/entity/Q39099310

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

@zh-cn

name

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@en

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@nl

type

label

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@en

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@nl

prefLabel

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@en

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@nl

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P356

J.CMET.2013.07.012

P1433

Cell Metabolism

P1476

Stat3 activation links a C/EBPδ to myostatin pathway to stimulate loss of muscle mass.

@en

P2093

David J Tweardy

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Jenny Pan

http://www.w3.org/2001/XMLSchema#string

Liping Zhang

http://www.w3.org/2001/XMLSchema#string

William E Mitch

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Yanjun Dong

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Yanlan Dong

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P2860

A nuclear factor for IL-6 expression (NF-IL6) is a member of a C/EBP family

Myostatin reduces Akt/TORC1/p70S6K signaling, inhibiting myoblast differentiation and myotube size

CCAAT/enhancer-binding proteins: structure, function and regulation

SOCS-1 and SOCS-3 block insulin signaling by ubiquitin-mediated degradation of IRS1 and IRS2

Protein degradation by the ubiquitin-proteasome pathway in normal and disease states

Cytokine signal transduction

Chemical probes that competitively and selectively inhibit Stat3 activation

C/EBP DNA-binding activity is upregulated by a glucocorticoid-dependent mechanism in septic muscle

Multiple types of skeletal muscle atrophy involve a common program of changes in gene expression

Myostatin and insulin-like growth factor-I and -II expression in the muscle of rats exposed to the microgravity environment of the NeuroLab space shuttle flight

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368-379

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scholarly article

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10.1016/J.CMET.2013.07.012

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3

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18

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Giacomo Garibotto

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2013-09-01T00:00:00Z

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256467080

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24011072

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3794464

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