Antiepileptics topiramate and levetiracetam alleviate behavioral deficits and reduce neuropathology in APPswe/PS1dE9 transgenic mice. - Wikidata - awgldk - TriplyDB

Antiepileptics topiramate and levetiracetam alleviate behavioral deficits and reduce neuropathology in APPswe/PS1dE9 transgenic mice.

Antiepileptics topiramate and levetiracetam alleviate behavioral deficits and reduce neuropathology in APPswe/PS1dE9 transgenic mice.

http://www.wikidata.org/entity/Q39119968

about

Drug repositioning approaches for the discovery of new therapeutics for Alzheimer's disease Brivaracetam, but not ethosuximide, reverses memory impairments in an Alzheimer's disease mouse model.Reproductive toxicity after levetiracetam administration in male rats: Evidence for role of hormonal status and oxidative stress.Nutritional and Pharmacological Strategies to Regulate Microglial Polarization in Cognitive Aging and Alzheimer's Disease.Response of the medial temporal lobe network in amnestic mild cognitive impairment to therapeutic intervention assessed by fMRI and memory task performance Signaling pathway cross talk in Alzheimer's disease GABAergic neurotransmission and new strategies of neuromodulation to compensate synaptic dysfunction in early stages of Alzheimer's disease.The vicious circle of hypometabolism in neurodegenerative diseases: Ways and mechanisms of metabolic correction.Treatment of epilepsy in patients with Alzheimer's disease.Reversal of high fat diet-induced obesity improves glucose tolerance, inflammatory response, β-amyloid accumulation and cognitive decline in the APP/PSEN1 mouse model of Alzheimer's disease.Epileptic activity in Alzheimer's disease: causes and clinical relevance.Incidence and impact of subclinical epileptiform activity in Alzheimer's disease.Targeting Neural Hyperactivity as a Treatment to Stem Progression of Late-Onset Alzheimer's Disease.Levetiracetam Affects Differentially Presynaptic Proteins in Rat Cerebral Cortex.Heightened cortical excitability in aged rodents with memory impairment.Cognition and dementia in older patients with epilepsy.Levetiracetam Alters Oscillatory Connectivity in Alzheimer's Disease.PM2.5 exposure aggravates oligomeric amyloid beta-induced neuronal injury and promotes NLRP3 inflammasome activation in an in vitro model of Alzheimer's disease.

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P2860

Antiepileptics topiramate and levetiracetam alleviate behavioral deficits and reduce neuropathology in APPswe/PS1dE9 transgenic mice.

http://www.wikidata.org/entity/Q39119968

description

2013 nî lūn-bûn

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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CNS Neuroscience & Therapeutics

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Antiepileptics topiramate and ...... APPswe/PS1dE9 transgenic mice.

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P2093

Bian-Rong Wang

http://www.w3.org/2001/XMLSchema#string

Hong-Guang Xie

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Jian-Quan Shi

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Li Gao

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Shu-Li Zhao

http://www.w3.org/2001/XMLSchema#string

Teng Jiang

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Ying-Dong Zhang

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You-Yong Tian

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P2860

An epigenetic blockade of cognitive functions in the neurodegenerating brain

Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.

'Clinical trials in Alzheimer's disease': immunotherapy approaches

Topiramate treatment is neuroprotective and reduces oligodendrocyte loss after cervical spinal cord injury

Mutant presenilins specifically elevate the levels of the 42 residue beta-amyloid peptide in vivo: evidence for augmentation of a 42-specific gamma secretase

Alzheimer's disease: the amyloid cascade hypothesis

Decreased clearance of CNS beta-amyloid in Alzheimer's disease

Aberrant excitatory neuronal activity and compensatory remodeling of inhibitory hippocampal circuits in mouse models of Alzheimer's disease

Chronic divalproex sodium use and brain atrophy in Alzheimer disease

Abnormal neuronal networks and seizure susceptibility in mice overexpressing the APP intracellular domain.

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10.1111/CNS.12144

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