Inducing apoptosis of cancer cells using small-molecule plant compounds that bind to GRP78.
about
The Natural Occurring Compounds Targeting Endoplasmic Reticulum StressAdaptive preconditioning in neurological diseases – therapeutic insights from proteostatic perturbationsEndoplasmic reticulum stress-mediated pathways to both apoptosis and autophagy: Significance for melanoma treatmentEndoplasmic reticulum chaperones and oxidoreductases: critical regulators of tumor cell survival and immunorecognitionBroad targeting of angiogenesis for cancer prevention and therapyEndoplasmic Reticulum Stress, Unfolded Protein Response, and Cancer Cell FateHKH40A downregulates GRP78/BiP expression in cancer cells.Epigallocatechin-3-gallate enhances ER stress-induced cancer cell apoptosis by directly targeting PARP16 activity.Endoplasmic reticulum stress and cancer.Proteomic analysis of differentially expressed proteins in 5-fluorouracil-treated human breast cancer MCF-7 cells.Expression and significance of glucose-regulated protein 78 in human osteosarcomaTargeting drivers of melanoma with synthetic small molecules and phytochemicals.Role of GRP78 in promoting therapeutic-resistant breast cancerIsochaihulactone-induced DDIT3 causes ER stress-PERK independent apoptosis in glioblastoma multiforme cells.Targeting the unfolded protein response in disease.Glucose-regulated proteins in cancer: molecular mechanisms and therapeutic potentialMolecular pathways: the PERKs and pitfalls of targeting the unfolded protein response in cancer.Honokiol suppresses metastasis of renal cell carcinoma by targeting KISS1/KISS1R signaling.Oncogenic BRAF induces chronic ER stress condition resulting in increased basal autophagy and apoptotic resistance of cutaneous melanoma.Involvement of ER stress and activation of apoptotic pathways in fisetin induced cytotoxicity in human melanoma.HSPA5 Gene encoding Hsp70 chaperone BiP in the endoplasmic reticulum.Molecular docking and molecular dynamics studies reveal structural basis of inhibition and selectivity of inhibitors EGCG and OSU-03012 toward glucose regulated protein-78 (GRP78) overexpressed in glioblastoma.Honokiol Decreases Lung Cancer Metastasis through Inhibition of the STAT3 Signaling Pathway.Glucose-regulated protein 78 substrate-binding domain alters its conformation upon EGCG inhibitor binding to nucleotide-binding domain: Molecular dynamics studies.
P2860
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P2860
Inducing apoptosis of cancer cells using small-molecule plant compounds that bind to GRP78.
description
2013 nî lūn-bûn
@nan
2013年の論文
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2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
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2013年论文
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name
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@en
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@nl
type
label
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@en
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@nl
prefLabel
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@en
Inducing apoptosis of cancer c ...... compounds that bind to GRP78.
@nl
P2093
P2860
P356
P1476
Inducing apoptosis of cancer c ...... t compounds that bind to GRP78
@en
P2093
A R Hawkins
J L Arbiser
M Y Bonner
P Thompson
P2860
P2888
P304
P356
10.1038/BJC.2013.325
P407
P577
2013-06-27T00:00:00Z