Impaired Amino Acid Transport at the Blood Brain Barrier Is a Cause of Autism Spectrum Disorder. - Wikidata - awgldk - TriplyDB

Impaired Amino Acid Transport at the Blood Brain Barrier Is a Cause of Autism Spectrum Disorder.

Impaired Amino Acid Transport at the Blood Brain Barrier Is a Cause of Autism Spectrum Disorder.

http://www.wikidata.org/entity/Q39135788

about

Commensal bacteria and essential amino acids control food choice behavior and reproduction Neurobiological bases of autism-epilepsy comorbidity: a focus on excitation/inhibition imbalance.Vascular heterogeneity and specialization in development and disease.Brain barriers and brain fluid research in 2016: advances, challenges and controversies.Organ-specific protection mediated by cooperation between vascular and epithelial barriers.Branched-Chain Amino Acid Negatively Regulates KLF15 Expression via PI3K-AKT Pathway.Glutamine Transport and Mitochondrial Metabolism in Cancer Cell Growth.Endothelial Cell Metabolism.Impaired amino acid metabolism in autism spectrum disorders.Advanced glycation endproducts, dityrosine and arginine transporter dysfunction in autism - a source of biomarkers for clinical diagnosis.Insights into the Structure, Function, and Ligand Discovery of the Large Neutral Amino Acid Transporter 1, LAT1."Immune Gate" of Psychopathology-The Role of Gut Derived Immune Activation in Major Psychiatric Disorders.Regulation of T3 Availability in the Developing Brain: The Mouse Genetics Contribution.Autism spectrum disorder: prospects for treatment using gene therapy.The Human SLC7A5 (LAT1): The Intriguing Histidine/Large Neutral Amino Acid Transporter and Its Relevance to Human Health.Clinical genetics and genomic medicine in Qatar Slc7a5 regulates Kv1.2 channels and modifies functional outcomes of epilepsy-linked channel mutations Elimination of substances from the brain parenchyma: efflux via perivascular pathways and via the blood-brain barrier Clinical and Molecular Characteristics of Mitochondrial Dysfunction in Autism Spectrum Disorder Preterm Birth and the Risk of Neurodevelopmental Disorders - Is There a Role for Epigenetic Dysregulation?Description of LAT1 Transport Mechanism at an Atomistic Level The Regulation and Function of the L-Type Amino Acid Transporter 1 (LAT1) in Cancer

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Impaired Amino Acid Transport at the Blood Brain Barrier Is a Cause of Autism Spectrum Disorder.

http://www.wikidata.org/entity/Q39135788

description

2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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name

Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

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Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

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type

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Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

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Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

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prefLabel

Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

@en

Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

@nl

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Impaired Amino Acid Transport ...... e of Autism Spectrum Disorder.

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Ahmet Okay Caglayan

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Anide Johansen

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Avner Schlessinger

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Cesare Indiveri

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Christoph P Dotter

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Dora C Tărlungeanu

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Elena Deliu

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Emanuela Morelli

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Fatma Mujgan Sonmez

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Gaia Novarino

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P2860

Amino-acid transport by heterodimers of 4F2hc/CD98 and members of a permease family

Sestrin2 is a leucine sensor for the mTORC1 pathway

Synaptic, transcriptional and chromatin genes disrupted in autism

Mutations in BCKD-kinase lead to a potentially treatable form of autism with epilepsy

Autism as a strongly genetic disorder: evidence from a British twin study

Regulated expression of ATF5 is required for the progression of neural progenitor cells to neurons

Neuroligin 2 is exclusively localized to inhibitory synapses

Translational control of hippocampal synaptic plasticity and memory by the eIF2alpha kinase GCN2

Autism-related deficits via dysregulated eIF4E-dependent translational control.

Histidinaemia: a benign metabolic disorder.

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1481-1494.e18

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Avner Schlessinger

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Autism spectrum

blood–brain barrier

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