Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1. - Wikidata - awgldk - TriplyDB

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1.

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1.

http://www.wikidata.org/entity/Q39143432

about

DAI/ZBP1 recruits RIP1 and RIP3 through RIP homotypic interaction motifs to activate NF-kappaB Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation Many stimuli pull the necrotic trigger, an overview Fueling the flames: Mammalian programmed necrosis in inflammatory diseases Photoreceptor cell death and rescue in retinal detachment and degenerations Viral infection and the evolution of caspase 8-regulated apoptotic and necrotic death pathways Necroptosis: The Trojan horse in cell autonomous antiviral host defense Receptor-interacting protein homotypic interaction motif-dependent control of NF-kappa B activation via the DNA-dependent activator of IFN regulatory factors Cooperative inhibition of RIP1-mediated NF-κB signaling by cytomegalovirus-encoded deubiquitinase and inactive homolog of cellular ribonucleotide reductase large subunit.The human cytomegalovirus UL36 gene controls caspase-dependent and -independent cell death programs activated by infection of monocytes differentiating to macrophages.The molecular regulation of programmed necrotic cell injury.Modulation of host innate and adaptive immune defenses by cytomegalovirus: timing is everything Pathogen-mediated proteolysis of the cell death regulator RIPK1 and the host defense modulator RIPK2 in human aortic endothelial cells.Ablation of the regulatory IE1 protein of murine cytomegalovirus alters in vivo pro-inflammatory TNF-alpha production during acute infection RIP kinases: key decision makers in cell death and innate immunity.The ribonucleotide reductase R1 subunits of herpes simplex virus 1 and 2 protect cells against poly(I · C)-induced apoptosis Suppression of RIP3-dependent necroptosis by human cytomegalovirus RIP1 Cleavage in the Kinase Domain Regulates TRAIL-Induced NF-κB Activation and Lymphoma Survival.Evidence for multiple cell death pathways during development of experimental cytomegalovirus retinitis in mice with retrovirus-induced immunosuppression: apoptosis, necroptosis, and pyroptosis Viral inhibition of BAK promotes murine cytomegalovirus dissemination to salivary glands.Two independent pathways of regulated necrosis mediate ischemia-reperfusion injury RIP3: a molecular switch for necrosis and inflammation.Toll-like receptor 3-mediated necrosis via TRIF, RIP3, and MLKL.Multifaceted evasion of the interferon response by cytomegalovirus.True grit: programmed necrosis in antiviral host defense, inflammation, and immunogenicity.A single viral gene determines lethal cross-species neurovirulence of baboon herpesvirus HVP2.Murine gammaherpesvirus 68 encodes a second PML-modifying protein.Going up in flames: necrotic cell injury and inflammatory diseases.Live or let die: manipulation of cellular suicide programs by murine cytomegalovirus.Interplay between CMVs and interferon signaling: implications for pathogenesis and therapeutic intervention.Prevention of cellular suicide by cytomegaloviruses.Tumour necrosis factor in infectious disease.Viral modulation of programmed necrosis.Necroptosis in immunity and ischemia-reperfusion injury.Regulation of RIP1 kinase signalling at the crossroads of inflammation and cell death.Ars Moriendi; the art of dying well - new insights into the molecular pathways of necroptotic cell death.Staying alive: cell death in antiviral immunity.Manipulation of apoptosis and necroptosis signaling by herpesviruses The diverse role of RIP kinases in necroptosis and inflammation.Global Screening of Antiviral Genes that Suppress Baculovirus Transgene Expression in Mammalian Cells.

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P2860

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein (RIP) homotypic interaction motif (RHIM)-dependent interaction with RIP1.

http://www.wikidata.org/entity/Q39143432

description

2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

@zh-cn

name

Cytomegalovirus M45 cell death ...... pendent interaction with RIP1.

@en

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein

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type

label

Cytomegalovirus M45 cell death ...... pendent interaction with RIP1.

@en

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein

@nl

prefLabel

Cytomegalovirus M45 cell death ...... pendent interaction with RIP1.

@en

Cytomegalovirus M45 cell death suppression requires receptor-interacting protein

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Journal of Biological Chemistry

P1476

Cytomegalovirus M45 cell death ...... pendent interaction with RIP1.

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P2093

Edward S Mocarski

http://www.w3.org/2001/XMLSchema#string

Jason W Upton

http://www.w3.org/2001/XMLSchema#string

William J Kaiser

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of RIP3, a RIP-like kinase that activates apoptosis and NFkappaB

RIP3, a novel apoptosis-inducing kinase

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

Toll-like receptors 9 and 3 as essential components of innate immune defense against mouse cytomegalovirus infection

Activation of death-inducing signaling complex (DISC) by pro-apoptotic C-terminal fragment of RIP

RIP1, a kinase on the crossroads of a cell's decision to live or die

Mouse receptor interacting protein 3 does not contain a caspase-recruiting or a death domain but induces apoptosis and activates NF-kappaB

Identification of Lps2 as a key transducer of MyD88-independent TIR signalling

P304

16966-16970

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scholarly article

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10.1074/JBC.C800051200

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25

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283

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2008-04-28T00:00:00Z

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18442983

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Cytomegalovirus

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