The induction of MIG6 under hypoxic conditions is critical for dormancy in primary cultured lung cancer cells with activating EGFR mutations.

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Radiosensitivity of quiescent and proliferating cells grown as multicellular tumor spheroids Tumor cell dormancy as an adaptive cell stress response mechanism.

P2860

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The induction of MIG6 under hypoxic conditions is critical for dormancy in primary cultured lung cancer cells with activating EGFR mutations.

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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The induction of MIG6 under hy ...... ith activating EGFR mutations.

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P1476

The induction of MIG6 under hy ...... ith activating EGFR mutations.

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P2093

F Imamura

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M Inoue

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P2860

Hallmarks of Cancer: The Next Generation

Gene 33/Mig-6, a transcriptionally inducible adapter protein that binds GTP-Cdc42 and activates SAPK/JNK. A potential marker transcript for chronic pathologic conditions, such as diabetic nephropathy. Possible role in the response to persistent stre

Acquired resistance of lung adenocarcinomas to gefitinib or erlotinib is associated with a second mutation in the EGFR kinase domain

Mechanisms of disseminated cancer cell dormancy: an awakening field

EGFR mutations in lung cancer: correlation with clinical response to gefitinib therapy.

MET amplification leads to gefitinib resistance in lung cancer by activating ERBB3 signaling.

Activating mutations in the epidermal growth factor receptor underlying responsiveness of non-small-cell lung cancer to gefitinib

Carbonic anhydrase IX expression, a novel surrogate marker of tumor hypoxia, is associated with a poor prognosis in non-small-cell lung cancer

Evidence that MIG-6 is a tumor-suppressor gene

Hypoxia promotes the differentiated human articular chondrocyte phenotype through SOX9-dependent and -independent pathways

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onc.2016.431

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2824-2834

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scholarly article

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10.1038/ONC.2016.431

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English

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P921

hypoxia

The induction of MIG6 under hypoxic conditions is critical for dormancy in primary cultured lung cancer cells with activating EGFR mutations.

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P2860

P2860

The induction of MIG6 under hypoxic conditions is critical for dormancy in primary cultured lung cancer cells with activating EGFR mutations.

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