An inquiry into the mechanisms of herpes simplex virus latency.
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Mutual Interference between Cytomegalovirus and Reconstitution of Protective Immunity after Hematopoietic Cell TransplantationThe role of the CoREST/REST repressor complex in herpes simplex virus 1 productive infection and in latencyHerpes simplex virus type 1 latency and reactivation: an updateA cultured affair: HSV latency and reactivation in neuronsDirect transfer of viral and cellular proteins from varicella-zoster virus-infected non-neuronal cells to human axonsHuman herpes viruses latent infection in the nervous systemThe Oct-2 transcription factor represses tyrosine hydroxylase expression via a heptamer TAATGARAT-like motif in the gene promoterHerpesviruses: latency and reactivation - viral strategies and host responseIntragenomic linear amplification of human herpesvirus 6B oriLyt suggests acquisition of oriLyt by transpositionMicrotubule-mediated transport of incoming herpes simplex virus 1 capsids to the nucleus.Persistent humoral immune responses in the CNS limit recovery of reactivated murine cytomegalovirusViral vectors for gene therapy in Parkinson's disease.Herpes simplex virus type 1 latency-associated transcripts are evidently not essential for latent infection.Regulation of DNA virus transcription by cellular POU family transcription factors.Interferon alpha induces establishment of alphaherpesvirus latency in sensory neurons in vitro.Preemptive CD8 T-cell immunotherapy of acute cytomegalovirus infection prevents lethal disease, limits the burden of latent viral genomes, and reduces the risk of virus recurrence.Long-term transgene expression in mice infected with a herpes simplex virus type 1 mutant severely impaired for immediate-early gene expression.Evidence for a bidirectional element located downstream from the herpes simplex virus type 1 latency-associated promoter that increases its activity during latencyGamma interferon (IFN-gamma) receptor null-mutant mice are more susceptible to herpes simplex virus type 1 infection than IFN-gamma ligand null-mutant miceViral vectors in the treatment of Parkinson's disease.Enhancer and long-term expression functions of herpes simplex virus type 1 latency-associated promoter are both located in the same regionRandom, asynchronous, and asymmetric transcriptional activity of enhancer-flanking major immediate-early genes ie1/3 and ie2 during murine cytomegalovirus latency in the lungs.Amplification by host cell factors of a sequence contained within the herpes simplex virus 1 genome.Herpes latency, meningitis, radiculomyelopathy and disseminated infection.A spread-deficient cytomegalovirus for assessment of first-target cells in vaccination.Murine gammaherpesvirus 68 LANA is essential for virus reactivation from splenocytes but not long-term carriage of viral genome.Use of differential display reverse transcription-PCR to reveal cellular changes during stimuli that result in herpes simplex virus type 1 reactivation from latency: upregulation of immediate-early cellular response genes TIS7, interferon, and interEvidence that herpes simplex virus DNA derived from quiescently infected cells in vitro, and latently infected cells in vivo, is physically damagedEntry of herpes simplex virus type 1 (HSV-1) into the distal axons of trigeminal neurons favors the onset of nonproductive, silent infectionHerpes simplex virus latent RNA (LAT) is not required for latent infection in the mouse.Recombinant mouse cytomegalovirus expressing a ligand for the NKG2D receptor is attenuated and has improved vaccine properties.Herpes simplex viruses.Gene therapy with herpes simplex virus vectors: progress and prospects for clinical neuroscience.Reverse genetics modification of cytomegalovirus antigenicity and immunogenicity by CD8 T-cell epitope deletion and insertion.Tegument protein control of latent herpesvirus establishment and animation.Transcription factors interacting with herpes simplex virus alpha gene promoters in sensory neuronsThe checkpoints of viral gene expression in productive and latent infection: the role of the HDAC/CoREST/LSD1/REST repressor complexSuppression of herpes simplex virus type 1 reactivation from latency by (+-)-9-([(Z)-2-(hydroxymethyl)cyclohexyl]methyl) guanine (L-653,180) in vitro.Adaptive and innate transforming growth factor beta signaling impact herpes simplex virus 1 latency and reactivationRole of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1
P2860
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P2860
An inquiry into the mechanisms of herpes simplex virus latency.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
An inquiry into the mechanisms of herpes simplex virus latency.
@en
An inquiry into the mechanisms of herpes simplex virus latency.
@nl
type
label
An inquiry into the mechanisms of herpes simplex virus latency.
@en
An inquiry into the mechanisms of herpes simplex virus latency.
@nl
prefLabel
An inquiry into the mechanisms of herpes simplex virus latency.
@en
An inquiry into the mechanisms of herpes simplex virus latency.
@nl
P1476
An inquiry into the mechanisms of herpes simplex virus latency.
@en
P2093
P304
P356
10.1146/ANNUREV.MI.41.100187.002551
P577
1987-01-01T00:00:00Z