Identifying targets for the restoration and reactivation of BRM.
about
The co-existence of transcriptional activator and transcriptional repressor MEF2 complexes influences tumor aggressiveness.RNAi screening identifies HAT1 as a potential drug target in esophageal squamous cell carcinoma.Flavonoids from each of the six structural groups reactivate BRM, a possible cofactor for the anticancer effects of flavonoidsThe silencing of the SWI/SNF subunit and anticancer gene BRM in Rhabdoid tumorsModulation of Brahma expression by the mitogen-activated protein kinase/extracellular signal regulated kinase pathway is associated with changes in melanoma proliferation.Expression inactivation of SMARCA4 by microRNAs in lung tumorsConcomitant loss of SMARCA2 and SMARCA4 expression in small cell carcinoma of the ovary, hypercalcemic typeThe multi-omic landscape of transcription factor inactivation in cancer.KAT5 and KAT6B are in positive regulation on cell proliferation of prostate cancer through PI3K-AKT signaling.Mechanism of BRG1 silencing in primary cancers.Selective class IIa HDAC inhibitors: myth or reality.Oncogenic targeting of BRM drives malignancy through C/EBPβ-dependent induction of α5 integrin.Beyond Mutations: Additional Mechanisms and Implications of SWI/SNF Complex Inactivation.Phospho-ΔNp63α/microRNA network modulates epigenetic regulatory enzymes in squamous cell carcinomasEpigenomic regulation of oncogenesis by chromatin remodeling.Myocyte enhancer factor 2D promotes tumorigenicity in malignant glioma cells.BRG1 and BRM loss selectively impacts RB and P53, respectively: BRG1 and BRM have differential functions in vivo.Frequent co-inactivation of the SWI/SNF subunits SMARCB1, SMARCA2 and PBRM1 in malignant rhabdoid tumours.Induction of functional Brm protein from Brm knockout miceLoss of the SWI/SNF ATPase subunits BRM and BRG1 drives lung cancer development.BRM Promoter Polymorphisms and Survival of Advanced Non-Small Cell Lung Cancer Patients in the Princess Margaret Cohort and CCTG BR.24 Trial.Two BRM promoter polymorphisms predict poor survival in patients with hepatocellular carcinoma.SMARCA4-deficient thoracic sarcoma: a distinctive clinicopathological entity with undifferentiated rhabdoid morphology and aggressive behavior.
P2860
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P2860
Identifying targets for the restoration and reactivation of BRM.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Identifying targets for the restoration and reactivation of BRM.
@en
Identifying targets for the restoration and reactivation of BRM.
@nl
type
label
Identifying targets for the restoration and reactivation of BRM.
@en
Identifying targets for the restoration and reactivation of BRM.
@nl
prefLabel
Identifying targets for the restoration and reactivation of BRM.
@en
Identifying targets for the restoration and reactivation of BRM.
@nl
P2093
P2860
P356
P1433
P1476
Identifying targets for the restoration and reactivation of BRM.
@en
P2093
P2860
P2888
P304
P356
10.1038/ONC.2012.613
P407
P577
2013-03-25T00:00:00Z
P5875
P6179
1018158675