Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

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Increasing autophagy and blocking Nrf2 suppress laminopathy-induced age-dependent cardiac dysfunction and shortened lifespan.The involvement of the nuclear lamina in human and rodent spermiogenesis: a systematic review.

P2860

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Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

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http://www.wikidata.org/entity/Q39181367

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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2017年论文

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2017年论文

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name

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@en

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@nl

type

Item

label

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@en

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

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prefLabel

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@en

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@nl

P1476

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

@en

P2093

Allyson Zabell

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W H Wilson Tang

http://www.w3.org/2001/XMLSchema#string

Wonshill Koh

http://www.w3.org/2001/XMLSchema#string

Xi Wang

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P2860

Mutations in the gene encoding lamin A/C cause autosomal dominant Emery-Dreifuss muscular dystrophy

Alteration of nuclear lamin organization inhibits RNA polymerase II-dependent transcription

Nuclear lamins A and B1: different pathways of assembly during nuclear envelope formation in living cells

Linear arrays of nuclear envelope proteins harness retrograde actin flow for nuclear movement.

Coupling of the nucleus and cytoplasm: role of the LINC complex

LINC complex alterations in DMD and EDMD/CMT fibroblasts

Mammalian SUN protein interaction networks at the inner nuclear membrane and their role in laminopathy disease processes

Loss of a DNA binding site within the tail of prelamin A contributes to altered heterochromatin anchorage by progerin

Inhibiting farnesylation of progerin prevents the characteristic nuclear blebbing of Hutchinson-Gilford progeria syndrome

Reversal of the cellular phenotype in the premature aging disease Hutchinson-Gilford progeria syndrome

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s11936-017-0520-z

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scholarly article

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10.1007/S11936-017-0520-Z

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Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

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P2860

P2860

Lamin A/C Cardiomyopathies: Current Understanding and Novel Treatment Strategies.

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P1433

P1476

P2093

P2860

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P1433

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