about
Reduced H3K27me3 and DNA hypomethylation are major drivers of gene expression in K27M mutant pediatric high-grade gliomasInternational network of cancer genome projects.Molecular subgroups of medulloblastoma: the current consensusK27M mutation in histone H3.3 defines clinically and biologically distinct subgroups of pediatric diffuse intrinsic pontine gliomasDissecting the genomic complexity underlying medulloblastomaMAPK pathway activation in pilocytic astrocytomaGenome sequencing of SHH medulloblastoma predicts genotype-related response to smoothened inhibitionOutcome prediction in pediatric medulloblastoma based on DNA copy-number aberrations of chromosomes 6q and 17q and the MYC and MYCN lociSubgroup-specific alternative splicing in medulloblastomaSubgroup-specific structural variation across 1,000 medulloblastoma genomesThe eEF2 kinase confers resistance to nutrient deprivation by blocking translation elongationFBW7 suppression leads to SOX9 stabilization and increased malignancy in medulloblastomaDriver mutations in histone H3.3 and chromatin remodelling genes in paediatric glioblastomaRole of LIM and SH3 protein 1 (LASP1) in the metastatic dissemination of medulloblastoma.Adaptor FYB (Fyn-binding protein) regulates integrin-mediated adhesion and mediator release: differential involvement of the FYB SH3 domainCancer Screening Recommendations for Individuals with Li-Fraumeni Syndrome.Early phase clinical trials of anticancer agents in children and adolescents - an ITCC perspective.Mechismo: predicting the mechanistic impact of mutations and modifications on molecular interactions.Assessing CpG island methylator phenotype, 1p/19q codeletion, and MGMT promoter methylation from epigenome-wide data in the biomarker cohort of the NOA-04 trial.Replicative senescence of mesenchymal stem cells: a continuous and organized processNuclear relocation of STAT6 reliably predicts NAB2-STAT6 fusion for the diagnosis of solitary fibrous tumour.Low physiologic oxygen tensions reduce proliferation and differentiation of human multipotent mesenchymal stromal cells.DNA methylation pattern changes upon long-term culture and aging of human mesenchymal stromal cellsPrognostic significance of clinical, histopathological, and molecular characteristics of medulloblastomas in the prospective HIT2000 multicenter clinical trial cohort.Pediatric high-grade glioma: biologically and clinically in need of new thinkingIntegrating Tenascin-C protein expression and 1q25 copy number status in pediatric intracranial ependymoma prognostication: A new model for risk stratification.The transcription factor evi-1 is overexpressed, promotes proliferation, and is prognostically unfavorable in infratentorial ependymomas.Embryonal tumor with abundant neuropil and true rosettes (ETANTR), ependymoblastoma, and medulloepithelioma share molecular similarity and comprise a single clinicopathological entity.Arhgap36-dependent activation of Gli transcription factors.Delineation of two clinically and molecularly distinct subgroups of posterior fossa ependymoma.Molecular subgroups of medulloblastoma: an international meta-analysis of transcriptome, genetic aberrations, and clinical data of WNT, SHH, Group 3, and Group 4 medulloblastomas.Phase II study of sorafenib in children with recurrent or progressive low-grade astrocytomas.FSTL5 is a marker of poor prognosis in non-WNT/non-SHH medulloblastoma.Epigenomic alterations define lethal CIMP-positive ependymomas of infancyGenome sequencing of pediatric medulloblastoma links catastrophic DNA rearrangements with TP53 mutations.DNA copy number alterations in central primitive neuroectodermal tumors and tumors of the pineal region: an international individual patient data meta-analysis.Recurrent somatic alterations of FGFR1 and NTRK2 in pilocytic astrocytoma.Enhancer hijacking activates GFI1 family oncogenes in medulloblastoma.Specific detection of methionine 27 mutation in histone 3 variants (H3K27M) in fixed tissue from high-grade astrocytomasIdentification of gains on 1q and epidermal growth factor receptor overexpression as independent prognostic markers in intracranial ependymoma.
P50
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P50
description
hulumtues
@sq
onderzoeker
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հետազոտող
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name
Stefan M Pfister
@ast
Stefan M Pfister
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Stefan M Pfister
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Stefan M Pfister
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Stefan M Pfister
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type
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Stefan M Pfister
@ast
Stefan M Pfister
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Stefan M Pfister
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Stefan M Pfister
@nl
Stefan M Pfister
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altLabel
Stefan Pfister
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prefLabel
Stefan M Pfister
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Stefan M Pfister
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Stefan M Pfister
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Stefan M Pfister
@nl
Stefan M Pfister
@sl
P214
P1053
F-6860-2013
P106
P21
P214
P31
P3829
P496
0000-0002-5447-5322
P734
P735
P7859
viaf-50145788