Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy - Wikidata - awgldk - TriplyDB

Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy

Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy

http://www.wikidata.org/entity/Q39210948

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Acute Hypoxia Induced an Imbalanced M1/M2 Activation of Microglia through NF-κB Signaling in Alzheimer's Disease Mice and Wild-Type Littermates.Antibiotic-induced perturbations in microbial diversity during post-natal development alters amyloid pathology in an aged APPSWE/PS1ΔE9 murine model of Alzheimer's disease.Autoimmunity as a Driving Force of Cognitive Evolution.The direct role of 5-lipoxygenase on tau pathology, synaptic integrity and cognition in a mouse model of tauopathy.Involvement of Astrocytes in Alzheimer's Disease from a Neuroinflammatory and Oxidative Stress Perspective.Overexpression of 5-Lipoxygenase Worsens the Phenotype of a Mouse Model of Tauopathy.Brain 5-lipoxygenase over-expression worsens memory, synaptic integrity, and tau pathology in the P301S mice.Functional and structural damage of neurons by innate immune mechanisms during neurodegeneration.Molecular and functional signatures in a novel Alzheimer's disease mouse model assessed by quantitative proteomics.Antileukotriene therapy by reducing tau phosphorylation improves synaptic integrity and cognition of P301S transgenic mice.Inflammation in CNS Neurodegenerative Diseases.Complex role of chemokine mediators in animal models of Alzheimer's Disease.Tau and neuroinflammation: What impact for Alzheimer's Disease and Tauopathies?[Tau and cognitive disorders: a role for T lymphocytes].Coconut (Cocos nucifera) Ethanolic Leaf Extract Reduces Amyloid-β (1-42) Aggregation and Paralysis Prevalence in Transgenic Caenorhabditis elegans Independently of Free Radical Scavenging and Acetylcholinesterase Inhibition.A Novel Association of Polymorphism in the ITGA4 Gene Encoding the VLA-4 α4 Subunit with Increased Risk of Alzheimer's Disease.PQQ ameliorates D-galactose induced cognitive impairments by reducing glutamate neurotoxicity via the GSK-3β/Akt signaling pathway in mouse.Therapeutic implications of how TNF links APOE, P-tau, α-synuclein, β-amyloid, and insulin resistance in neurodegenerative diseases Reinstating plasticity and memory in a tauopathy mouse model with an acetyltransferase activator Microglia prevent peripheral immune cell invasion and promote an anti-inflammatory environment in the brain of APP-PS1 transgenic mice A Closer Look into the Role of Protein Tau in the Identification of Promising Therapeutic Targets for Alzheimer's Disease

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Hippocampal T cell infiltration promotes neuroinflammation and cognitive decline in a mouse model of tauopathy

http://www.wikidata.org/entity/Q39210948

description

2016 nî lūn-bûn

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2016年論文

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2016年論文

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2016年论文

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2016年论文

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2016年论文

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Hippocampal T cell infiltratio ...... in a mouse model of tauopathy

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Hippocampal T cell infiltratio ...... in a mouse model of tauopathy.

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Hippocampal T cell infiltratio ...... in a mouse model of tauopathy

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Hippocampal T cell infiltratio ...... in a mouse model of tauopathy

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Hippocampal T cell infiltratio ...... in a mouse model of tauopathy.

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Dominique Demeyer

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Elodie Martin

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P2860

TREM2 Variants in Alzheimer's Disease

Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease

Amyloid plaque core protein in Alzheimer disease and Down syndrome

Role of Immediate-Early Genes in Synaptic Plasticity and Neuronal Ensembles Underlying the Memory Trace

Complement regulation of T cell immunity

Immune dysregulation and cognitive vulnerability in the aging brain: Interactions of microglia, IL-1β, BDNF and synaptic plasticity

Depletion of microglia and inhibition of exosome synthesis halt tau propagation.

Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model

Neuroinflammation in Alzheimer's disease

Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease

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Guillaume Dorothee

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