Helicobacter pylori activate epidermal growth factor receptor- and phosphatidylinositol 3-OH kinase-dependent Akt and glycogen synthase kinase 3beta phosphorylation.
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Helicobacter pylori-induced activation of beta-catenin involves low density lipoprotein receptor-related protein 6 and DishevelledMechanisms of disease: Helicobacter pylori virulence factorsWhat exists beyond cagA and vacA? Helicobacter pylori genes in gastric diseasesWnt/β-catenin, an oncogenic pathway targeted by H. pylori in gastric carcinogenesisHelicobacter pylori virulence and cancer pathogenesisHelicobacter pylori infection, oncogenic pathways and epigenetic mechanisms in gastric carcinogenesis.Phylogeographic origin of Helicobacter pylori determines host-adaptive responses upon coculture with gastric epithelial cells.Regulation of p53 tumor suppressor by Helicobacter pylori in gastric epithelial cells.Helicobacter pylori and gastric cancer: factors that modulate disease risk.VCP phosphorylation-dependent interaction partners prevent apoptosis in Helicobacter pylori-infected gastric epithelial cellsActivation of EGFR-PI3K-AKT signaling is required for Mycoplasma hyorhinis-promoted gastric cancer cell migration.H. pylori CagL-Y58/E59 prime higher integrin α5β1 in adverse pH condition to enhance hypochlorhydria vicious cycle for gastric carcinogenesis.Clinical relevance of cagPAI intactness in Helicobacter pylori isolates from Vietnam.Paxillin is a novel cellular target for converging Helicobacter pylori-induced cellular signaling.Promotion of cytoplasmic mislocalization of p27 by Helicobacter pylori in gastric cancer.Association of MTOR and AKT Gene Polymorphisms with Susceptibility and Survival of Gastric CancerHelicobacter pylori-associated regulation of forkhead transcription factors FoxO1/3a in human gastric cellsPathogenesis of Helicobacter pylori-Related Gastroduodenal Diseases from Molecular Epidemiological Studies.Helicobacter pylori CagA Suppresses Apoptosis through Activation of AKT in a Nontransformed Epithelial Cell Model of Glandular Acini Formation.Endotoxin induces proliferation of NSCLC in vitro and in vivo: role of COX-2 and EGFR activation.The significance of virulence factors in Helicobacter pylori.The Effect of Helicobacter pylori on Epidermal Growth Factor Receptor-Induced Signal Transduction and the Preventive Effect of Celecoxib in Gastric Cancer CellsH. pylori virulence factors: influence on immune system and pathology.Helicobacter pylori Induced Phosphatidylinositol-3-OH Kinase/mTOR Activation Increases Hypoxia Inducible Factor-1α to Promote Loss of Cyclin D1 and G0/G1 Cell Cycle Arrest in Human Gastric Cells.Adherens junctions as targets of microorganisms: a focus on Helicobacter pylori.Epigenetic modifications induced by Helicobacter pylori infection through a direct microbe-gastric epithelial cells cross-talk.The functional interplay of Helicobacter pylori factors with gastric epithelial cells induces a multi-step process in pathogenesis.Helicobacter pylori infection: host immune response, implications on gene expression and microRNAs.Role of epidermal growth factor receptor signaling in the interaction of Neisseria meningitidis with endothelial cells.Helicobacter pylori virulence factor CagA promotes tumorigenesis of gastric cancer via multiple signaling pathways.Expressional profiles of transcription factors in the progression of Helicobacter pylori-associated gastric carcinoma based on protein/DNA array analysis.STAT3 activation in infection and infection-associated cancer.Braf, Kras and Helicobacter pylori epigenetic changes-associated chronic gastritis in Egyptian patients with and without gastric cancer.Helicobacter pylori induces promoter hypermethylation and downregulates gene expression of IRX1 transcription factor on human gastric mucosa.Suppression of CD74 expression and Helicobacter pylori adhesion by auraptene targeting serum starvation-activated ERK1/2 in NCI-N87 gastric carcinoma cells.Helicobacter pylori CagA Protein Negatively Regulates Autophagy and Promotes Inflammatory Response via c-Met-PI3K/Akt-mTOR Signaling Pathway.A Tale of Two Toxins: Helicobacter Pylori CagA and VacA Modulate Host Pathways that Impact Disease.Effects of EGFR Inhibitor on Helicobacter pylori Induced Gastric Epithelial Pathology in Vivo.Protein kinase C isozymes regulate matrix metalloproteinase-1 expression and cell invasion in Helicobacter pylori infection.Helicobacter pylori CagA protein activates Akt and attenuates chemotherapeutics-induced apoptosis in gastric cancer cells.
P2860
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P2860
Helicobacter pylori activate epidermal growth factor receptor- and phosphatidylinositol 3-OH kinase-dependent Akt and glycogen synthase kinase 3beta phosphorylation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@en
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@nl
type
label
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@en
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@nl
prefLabel
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@en
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@nl
P2860
P1476
Helicobacter pylori activate e ...... kinase 3beta phosphorylation.
@en
P2093
Fazal H Tabassam
Yoshio Yamaoka
P2860
P356
10.1111/J.1462-5822.2008.01237.X
P577
2008-09-08T00:00:00Z