Reactive oxygen species are induced by Kaposi's sarcoma-associated herpesvirus early during primary infection of endothelial cells to promote virus entry.
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KSHV Entry and Trafficking in Target Cells-Hijacking of Cell Signal Pathways, Actin and Membrane DynamicsKaposi's sarcoma-associated herpesvirus induces Nrf2 activation in latently infected endothelial cells through SQSTM1 phosphorylation and interaction with polyubiquitinated Keap1The Polymorphisms with Cataract Susceptibility Impair the EPHA2 Receptor Stability and Its Cytoprotective FunctionKaposi's sarcoma-associated herpesvirus induces Nrf2 during de novo infection of endothelial cells to create a microenvironment conducive to infectionKaposi's sarcoma-associated herpesvirus genome programming during the early stages of primary infection of peripheral blood mononuclear cells.Lipoteichoic acid (LTA) and lipopolysaccharides (LPS) from periodontal pathogenic bacteria facilitate oncogenic herpesvirus infection within primary oral cellsEffect of total flavonoids of Spatholobus suberectus Dunn on PCV2 induced oxidative stress in RAW264.7 cells.The Kaposi's sarcoma-associated herpesvirus (KSHV)-induced 5-lipoxygenase-leukotriene B4 cascade plays key roles in KSHV latency, monocyte recruitment, and lipogenesis.Multiple Functions of Glutamate Uptake via Meningococcal GltT-GltM L-Glutamate ABC Transporter in Neisseria meningitidis Internalization into Human Brain Microvascular Endothelial CellsHPV16 E6 and E7 proteins induce a chronic oxidative stress response via NOX2 that causes genomic instability and increased susceptibility to DNA damage in head and neck cancer cells.Nuclear Innate Immune DNA Sensor IFI16 Is Degraded during Lytic Reactivation of Kaposi's Sarcoma-Associated Herpesvirus (KSHV): Role of IFI16 in Maintenance of KSHV Latency.High Glucose Induces Reactivation of Latent Kaposi's Sarcoma-Associated HerpesvirusRedox Regulation and the Autistic Spectrum: Role of Tryptophan Catabolites, Immuno-inflammation, Autoimmunity and the Amygdala.Rho'ing in and out of cells: viral interactions with Rho GTPase signalingInteraction of KSHV with host cell surface receptors and cell entry.Kaposi's sarcoma-associated herpesvirus induces the ATM and H2AX DNA damage response early during de novo infection of primary endothelial cells, which play roles in latency establishment.The Role of Human Herpesvirus 8 in Diabetes Mellitus Type 2: State of the Art and a Medical Hypothesis.Modulation of host ROS metabolism is essential for viral infection of a bloom-forming coccolithophore in the ocean.Oxidative stress enables Epstein-Barr virus-induced B-cell transformation by posttranscriptional regulation of viral and cellular growth-promoting factors.Commentary: High Glucose Induces Reactivation of Latent Kaposi's Sarcoma-Associated Herpesvirus.Oxeiptosis, a ROS-induced caspase-independent apoptosis-like cell-death pathway.Extracellular vesicles from KSHV-infected endothelial cells activate the complement system.
P2860
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P2860
Reactive oxygen species are induced by Kaposi's sarcoma-associated herpesvirus early during primary infection of endothelial cells to promote virus entry.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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name
Reactive oxygen species are in ...... cells to promote virus entry.
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Reactive oxygen species are in ...... cells to promote virus entry.
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type
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Reactive oxygen species are in ...... cells to promote virus entry.
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Reactive oxygen species are in ...... cells to promote virus entry.
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Reactive oxygen species are in ...... cells to promote virus entry.
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Reactive oxygen species are in ...... cells to promote virus entry.
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P2860
P356
P1433
P1476
Reactive oxygen species are in ...... l cells to promote virus entry
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P2093
Bala Chandran
Virginie Bottero
P2860
P304
P356
10.1128/JVI.02958-12
P407
P577
2012-11-21T00:00:00Z