Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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Recent advances in understanding spondyloarthritis.

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P1476

Recent advances in understanding spondyloarthritis

@en

P2093

J S H Gaston

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P2860

Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility.

Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci

ERAP1 reduces accumulation of aberrant and disulfide-linked forms of HLA-B27 on the cell surface

Endoplasmic reticulum stress-induced transcription factor, CHOP, is crucial for dendritic cell IL-23 expression

Functionally distinct ERAP1 allotype combinations distinguish individuals with Ankylosing Spondylitis.

Silencing or inhibition of endoplasmic reticulum aminopeptidase 1 (ERAP1) suppresses free heavy chain expression and Th17 responses in ankylosing spondylitis.

T-bet is a key modulator of IL-23-driven pathogenic CD4(+) T cell responses in the intestine.

Hallmarks of Tissue-Resident Lymphocytes.

ERAP1 association with ankylosing spondylitis is attributable to common genotypes rather than rare haplotype combinations.

IgA-coated E. coli enriched in Crohn's disease spondyloarthritis promote TH17-dependent inflammation.

P304

304

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P31

scholarly article

P356

10.12688/F1000RESEARCH.10739.1

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P478

http://www.w3.org/2001/XMLSchema#string

P50

J S H Gaston

P577

2017-03-22T00:00:00Z

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P698

28413624

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P932

5365215

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